The Death of Peers and Parents Should Change the Way We Live

Last week, I flew to Montreal to be with my mom in the last days before her death. While it was hard to see how much she had changed, it was sobering to be reminded of something I started to realize six years ago — that the death of friends and family should change the way we live.

The untimely death of two college friends in 2017 was the impetus for me to finally change my own diet and lifestyle. One of my friends died of a stroke and the other of a massive heart attack and both worked in healthcare their entire lives. As mentioned several times in “A Dietitian’s Journey,” I knew that my death would be next if I didn’t lose weight, lower my very high blood pressure and blood sugar.

While it took me twice as long as it should have to accomplish my health and weight goals due to undiagnosed hypothyroidism, I was successful in losing 55 pounds and taking a foot off my waist, and in putting my hypertension and type 2 diabetes into remission. Many times I was asked why I took accomplishing my goal so seriously and my reply was always the same, “I am doing this as if my life depends on it, because it does.”

When my dad was diagnosed with Alzheimer’s disease, I once again made some lifestyle changes. Even though I had put my type 2 diabetes into remission with diet, I began taking a low dose of the prescription medication Metformin preventatively, while continuing to eat a low carb diet. But, like many people, I became somewhat complacent and maybe even a little bit smug that diet alone was enough, and in the years since my dad’s death ended up discontinuing my medication, with my doctor’s knowledge.

Something that I was missing in my decision to discontinue this medication that I took prophylactically was that my health had changed, and I didn’t know it yet.

When I was diagnosed with profound hypothyroidism a little over a year ago, my doctor told me that even with taking thyroid hormone replacement medication that it would take a year and a half to fully recover due to how advanced it was. 

I wanted to understand how my body had changed, so I did lots of reading in the scientific literature and learned about how hypothyroidism affected my heart rate, my blood pressure and cholesterol and wrote about it here. For a while I took a “baby dose” of blood pressure medication and made hypothyroid-specific dietary changes but eventually stopped taking it, and waited for the thyroid medication to reverse the symptoms. In retrospect, that may have been a bit naive. I was assuming these metabolic markers would return to normal in time, thinking that all I needed to do was wait the full year and a half to fully recover. 

Recently, after an increase in thyroid medication I noticed that my blood sugar was significantly higher than it had been in years even though I had been compliant eating a low carb diet. Once again, I turned to the scientific literature to determine why. It was then that I discovered that all thyroid hormone replacement — even the “natural ones” raise blood sugar, and I wrote about that here

Given my higher blood sugar and my late dad’s diagnosis of Alzheimer’s disease, I started back on the half dose of metformin, but it wasn’t until my recent visit with my mom, that I became less complacent. 

Around the time my dad was diagnosed with Alzheimer’s, my mom was diagnosed with vascular dementia secondary to some mild strokes, known as TIAs. At first, the signs were subtle — things like difficulty organizing things she wanted to do into a list, but over the past few years, she lost the ability to read and write, and sometimes couldn’t organize her thoughts into coherent sentences. My mom didn’t have high blood pressure, but struggled her whole life with being overweight and was sedentary.

The important things for me — and that I wasn’t factoring in before was that my mom having had mini strokes, and later developing vascular dementia put me at risk because I now have moderate hypertension as a result of my thyroid condition. As well, my dad having had type 2 diabetes and developing Alzheimer’s disease increased my risk now that the thyroid medication wasn’t keeping my blood sugar in the non-diabetic range, as it had been for two years with diet alone.

It’s all nice and fine that I continue to eat low carb and workout at the gym, but in light of my thyroid meds raising my blood sugar, taking Metformin only makes sense. Likewise, it’s great that I achieved remission from hypertension with diet alone, but things have changed. My blood pressure has been moderately elevated since last year and discontinuing the low dose blood pressure medication hoping improved thyroid levels will normalize them is a bit naive. They may, but they may not.

While I was away in Montreal visiting with my mom, I made a phone appointment with my doctor. I told him about my mother’s diagnosis and my current blood pressure, and said I think it makes sense to take some blood pressure medication and to monitor it regularly. I also told him that I think given my blood sugar levels are higher even though I continue to eat low carb, that I think it only makes sense to begin taking Metformin again, and continue to monitor them regularly. He agreed.

Today I buried my mom.

While she died due to pneumonia and not vascular dementia, her death has changed how I will live. I realize that I can no longer be complacent that eating a good diet, and going to the gym several days a week is “enough.”

My dad is buried beside my mom and visiting his grave reminded me in a fresh way that his death was related to him having Alzheimer’s disease, and that he had type 2 diabetes for the last 40 years of his life. While my elevated blood sugar is a side-effect of the thyroid hormone medication that I have to take, taking Metformin and continuing to eat a low carb diet and exercising only makes sense. His death has changed how I will live. 

Final Thoughts

Dietary and lifestyle changes are very important and can effectively put both type 2 diabetes and hypertension into remission, however when circumstances change, it is necessary to consider medication as an adjunct. 

I have no choice but to be on thyroid medication in the same way that someone with type 1 diabetes has to take insulin. Given my lack of thyroid function, as well the side effects of taking thyroid hormones, I have chosen to let my parent’s deaths change the way I live. It may not be forever, and it may be — only time will tell. 

In the meantime, I will continue to eat a low carb diet to control my blood sugar as best I can, and to go to the gym several times a week to lift weights and do resistance training.

Taking medication is not a “failure”. Dying an unnecessary or premature death like my two girlfriends did, is.

And if taking medication, in addition to eating a good diet and being active helps avoid, or significantly slow dementia, all the better. 

To your good health, 

Joy

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Copyright ©2023 BetterByDesign Nutrition Ltd.

 

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

 

Success is All About “Showing Up”

Whether it’s achieving significant weight loss or recovering one’s health, I believe that success is all about “showing up”.  As much as people would like to believe that eating certain foods or taking supplements will cause the “pounds to melt away” or for the symptoms of a health condition to “improve in just 6 weeks,” we know better.  Success requires making a commitment, consistency and “showing up” for yourself — if not every time, most times.

One of the first posts I made when I started my personal weight-loss and health-recovery story (March 5, 2017 – March 4, 2019) was a video of me walking around a track.  I was so overweight and out of shape that I could barely walk and talk at the same time. I was clearly out of breath!  So why on earth did I post it? Why didn’t I wait until I lost the 55 pounds and the foot off my waist and then post my success pictures? I believe that people relate to the struggle of watching someone doing it and that watching them succeed no matter how long it takes, gives them hope that they can, too! 

Here’s a clip from that video;

What I said in that video still rings true today, as I am continuing to work towards recovering from severe hypothyroidism;

“I want to encourage people that if they make a commitment to “do it” just do it! If you have to re-schedule other things to do it, just do it! It’s important. Exercise is a small component of this journey, but it is an important component. “

It’s about “showing up”.

By setting goals and by being intentional about following through, I went from looking like the photo on the left to being able to hike intermediate trails, and looking like the photo on the right. By “showing up” for myself in terms of my eating, I was able to put my extremely high blood pressure and type 2 diabetes into remission. But losing 55 pounds and a foot off my waist didn’t happen overnight. Even though I am a Dietitian and have helped many others achieve this in far less time, as my doctor told me a year ago, there indications that I probably had subclinical hypothyroidism for the past 10 years. In a sense, that I was able to accomplish the weight loss and for the most part maintain it, is evidence to the power of “showing up.”

Achieving and maintaining a healthy body weight has really about me making decisions on a consistent basis that move me towards that goal. 

It’s not about being “perfect,” but about being consistent.

It’s about “showing up” for myself when I plan my meals. It’s about prioritizing foods that will provide satiety (not feeling hungry), and ensuring that the foods I eat with them provide me with the nutrients I need, while supporting my ongoing metabolic health.

Setbacks are About “Showing Up”

Weight loss and health recovery rarely occur in a straight line. There are any number of setbacks that can, and do occur. It is what we do with setbacks that determines whether we regain all the weight we lost and then some, or becomes as sick or sicker than we used to be. 

It is still about “showing up”.

At my youngest son’s wedding last June, I was at my sickest but didn’t yet have a diagnosis.  I could barely walk or get up from a chair because of the edema in my legs, and my tongue was so swollen that it was difficult to talk. You can read more about that in this first post in this new series

What made it really hard for me was that I looked like I did when I was 55 pounds heavier, but I wasn’t. In short, I looked like a “failure.”

Recovery from Hypothyroidism is About “Showing Up”

I wrote a few posts about my recovery from hypothyroidism under “A Dietitian’s Journey (Part II)” but there were a lot of “silent” periods in between those posts, and since the last one where I simply had to “show up.”

I needed to continue to take my thyroid meds and have my thyroid hormones checked every few months. I needed to continue to work with my doctor to get my medication adjusted to a level that enabled me to feel well, and to accomplish what I needed to.  I am very thankful to have an incredibly knowledgeable and supportive doctor, but the bottom line is that I need to consistently “show up.”

I needed to prepare my meds each evening for the following days dosages I take them in — and it didn’t matter if I was tired.

I had to continue to “show up” for myself when it came to making decisions as to which foods I will eat and those I choose not to because I have Hashimoto’s disease, which is an autoimmune condition.

I needed to continue to “show up” for myself to get tested for nutrients of importance in hypothyroidism and to supplement accordingly.  Except for vitamin D3 and K2, and later magnesium, I never took supplements — choosing instead to get them by eating a range of real, whole foods.  Being diagnosed with an autoimmune condition necessitated me changing my mind on this and consistently “showing up” for myself by consistently taking the supplements that I needed to.

Every day with hypothyroidism is about “showing up.”

Restoring Health is About “Showing Up”

A few weeks ago, after my new thyroid medications took effect and it became possible to think about regaining my mobility and muscle mass, the question was “how.” 

My son who had been my “hiking buddy” recently married and moved out of province, and I was very aware that I wasn’t ready to re-join the ladies hiking group that I am a member of without regaining my strength.  I needed a plan.

Someone in the local Facebook group posted about a program available and I knew that was exactly what I needed.  It was close and would give me access to the weight-training facilities that I needed to rebuild my muscles, and the classes would provide me with some fun and aerobic exercise at the same time. They even had several pools where I could to rekindle my love of swimming, after I replaced my size 16 bathing suit. 

In order to “show up” I had to first get what I needed.

By the end last weekend, I had a 3-month renewable membership to the fitness center, a one year parking pass, a new gym bag, and a bathing suit in my size. While I wondered if I would even remember how to swim as it had been at least 30 years since I last did, I was going to be focusing on doing what I needed to do to rebuild my muscle mass, and that was to “eat sufficient protein at each of my meals” and to “lift, push and pull heavy things often“.  

I planned to go to the gym for the first time this past Wednesday, but make sure there was nothing slowing me down to “showing up”, my gym bag was already packed, and by the front door. 

My gym socks and training shoes were pulled out, and my sweat band was draped on top of them.

My driver’s license, debit card and health card were already in a small wallet attached to my keys and was laying on top of my gym bag.

My water bottle was ready to go, and I had the coins needed for the lockers.

I had no excuses.

As I grabbed my bag and water bottle, I was “ready” to “show up” for myself. I needed to, because there was no one there to coax or encourage me. I had to be there for “me” and I was.

During my first workout, I learned how to use one of the types resistance machines and did three reps of ten with a 45 second rest in between each set on most of them — or as much as I could. I focused on having good form and didn’t try to do more than my body was able to do.  This was the first workout, and I was already successful because I “showed up”. Everything else I did was moving me towards my goal of getting in good enough shape to hike again.

Yesterday, I woke up quite sore — especially my pecs. These are not muscles I use much, if at all. I had thought I might go swimming, but I was so sore I wasn’t sure that was such a good idea since it had been way too long since I last did it. I stretched my muscles and rested and prepared my gym bag to go on Friday.

When I awoke today, everything was prepared. The only thing I grabbed was a mask because of the smoke warning outside, and headed to the gym.

I had already determined I would do what I could, given I am new at it and still somewhat sore from Wednesday, and had a good workout. I was surprised that I could increase weight or reps on some machines but definitely did less on the pec press.  That’s okay. I “showed up.”

Final Thoughts…

Whether it is losing significant weight, restoring one’s metabolic health, or rebuilding one’s physical strength after an illness, the first thing that is necessary is to make a commitment to “do it”.

Then, put the pieces in place that are required to be successful. If that is weight loss or health recovery, have a plan of how you are going to do it and who will oversee your progress. If it’s rebuilding your strength after an illness, find something that you want to do, that’s realistic for you to consistently do, and then get whatever is needed to get started.

Then, as I said in the video above, once you’ve made the commitment to “do it,” just do it.

If you are consistent in “showing up” for yourself on a regular basis, you will be successful in achieving your goals. 

To your good health,

Joy

You can follow me on:

Twitter: https://twitter.com/lchfRD
Facebook: https://www.facebook.com/BetterByDesignNutrition/

 

Copyright ©2023 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

 

A Dietitian’s Journey (Part III) – the next phase

Thirteen months ago, I was diagnosed with an auto-immune condition and was the sickest I had been in many years. Thankfully, I am entering the next phase, where I hope to restore my health to what it was before my diagnosis.

At the time of my youngest son’s wedding on June 3, 2022, I was incredibly swollen all over, had pronounced joint pain and muscle aches, and significant edema in my legs and ankles — so much so, that I was wearing compression stockings all the time, even at the wedding.

The skin on my cheeks was dry and  flaky and despite trying multiple types of intense moisturizers, nothing helped.  My mouth symptoms had progressed to the point that I found it difficult to say certain words because my tongue was swollen so much that it seemed too large for my mouth, and the salivary glands underneath my tongue were swollen.

The debilitating fatigue made life very difficult.

The muscle weakness had progressed to the point where it was difficult for me to get up from a chair, or to get out of the car without pushing myself up with my hands. My eldest son was helping me get to and from the beach for the photos for the wedding and he thought it was me aging. Even my other two sons began to assume the same thing because the changes occurred too slowly for them to see that they only started a few months ago.  I was thinking that I may have some form of “long-Covid,” but what got me starting to think that my symptoms may have had something to do with my thyroid was the very noticeable swelling in my face.

The phase immediately prior to diagnosis was the most difficult because on the occasion of my first son’s wedding, I looked like I did when I was 55 pounds heavier, but wasn’t. What was very stressful was not knowing why I was so sick.

LEFT: March 5, 2017, RIGHT: June 3, 2022 at my youngest son’s wedding.

The First Phase of Recovering from Hypothyroidism 

Just as weight loss and metabolic health recovery rarely take place in a straight line, the same is true with recovering from hypothyroidism.

At the end of October 2022, my thyroid hormones had improved significantly taking two types of thyroid medication. My free T3 (the active thyroid hormone) was at 65.5% of the reference range, however my free T4 was only at 41.75%.  Unfortunately, the medication adjustment we made at that time to “tweak things” made everything worse. By April, I was exhausted and cold, and found it very difficult to get through the day.

After having my thyroid hormones retested, it was evident why I felt awful, as my free T4 and free T3 had both dropped significantly.

Once again, my doctor adjusted my medication and thankfully my symptoms improved within a few weeks. Unfortunately, the less-than-optimal dose of thyroid hormones from January to April triggered another round of excessive hair shedding, but rather than “sweat the small stuff”, mid-June I opted for a new haircut which allowed my natural curls to fall where they may and it helped.

Since my condition is autoimmune, I continue to follow the specific dietary recommendations that I outlined here, as well as supplementing with the nutrients of importance in hypothyroidism while periodically having my serum nutrient levels tested.  I also continue to eat a lower carb diet that is high in highly bioavailable protein to support my continued metabolic health.

A Dietitian’s Journey Continues – the next phase

While I was feeling much better for the past few weeks I was bemoaning the fact that I was not well enough to resume hiking. I know, “first world problems”. Now that the son that got into hiking with me a few years ago was married and had moved out of town, it was up to me to figure out a way to regain my strength.

Last week, someone on social media posted about a local fitness program and I thought to myself, “I am going to join that“! It was perfect. It was close and would give me access to the weight-training facilities that I needed to rebuild my muscles, as well as classes that would provide me with some fun and aerobic exercise at the same time. They even had several pools where I could to re-discover my age-old love of swimming!  Realizing that the last bathing suit I owned was size 16 (from before I lost weight in 2017-2019), it was time to go shopping.

By the end of this past weekend, I already had a 3-month renewable fitness center membership, a one year parking pass, a new gym bag, and a bathing suit in my size. While I wondered to myself if I will even remember how to swim, but that was okay as I was determined to begin with “lifting, pushing and pulling heavy things often“.  Along with eating sufficient protein intake over each of my meals, weights and resistance training was the most important for restoring my muscle mass and avoiding sarcopenia (muscle-loss) so common in older adults. I was determined NOT to become one of those frail older people and this program would get me there.

A Dietitian’s Journey Continues

I was planning to start at the gym this past Tuesday but ended up taking one of my sons and daughter-in-laws to the airport. When I woke up on Wednesday morning, I was ready to go.  Not only was I ready, but I was excited to go, because for the first time in a year and a half to two years, it was possible. I was finally well enough again.

When I arrived at the fitness room, I asked the trainer to show me how to adjust the machines. She ushered me over to the cardio equipment, while encouraging me that this is where I should start.  I replied to her that my first priority is to rebuild lost muscle mass and asked her to please show me how to use the resistance equipment, which she did.  While cardio definitely has a place in my long-term goal of being able to hike again, lifting weights and doing resistance training will enable me to rebuild lost muscle, and strengthen joints and connective tissue as well as help with restoring my metabolism and mobilizing residual fat.

This is me after my first weight training session yesterday. I was sweaty and tired, but it felt amazing to finally be at this next phase of my journey one I have waited for as patiently as possible.

I hope that a Dietitian’s Journey – Part III encourages others who are recovering from Hashimoto’s as well as other types of auto-immune disorders.

If you would like more information about how I can support you in your health journey, please reach out to me through the Contact Me form on the tab above.

To your good health,

Joy

 

You can follow me on:

Twitter: https://twitter.com/lchfRD
Facebook: https://www.facebook.com/BetterByDesignNutrition/

 

Copyright ©2023 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

 

Thyroid Medication can Worsen Blood Sugar Control in People with Diabetes

Did you know that thyroid medication can worsen blood sugar? The ‘highlights of prescribing information” sheets available to pharmacists and doctors for medications such as Synthroid® (generic: levothyroxine) and Cytomel® (generic liothyronine) warn that both these types of thyroid medication can worsen blood sugar control in diabetics, and increase the need for diabetes medications, including insulin. 

I didn’t know this.

Even though I had been diagnosed with type 2 diabetes more than twelve years ago and with hypothyroidism this past August, when I was prescribed thyroid medication, the pharmacist didn’t mention it when I first filled my prescription, nor was I provided with any printed information when my thyroid medication was delivered. My doctor didn’t mention it either but understandably, he knew I had been in remission of diabetes for three years prior to be diagnosed with hypothyroidism and probably didn’t think of me as being diabetic.  He was well-aware that for three years prior to be diagnosed with hypothyroidism, I did not meet the diagnostic criteria for type 2 diabetes either on the basis of fasting blood glucose or HbA1C because I controlled my blood sugar through diet.

When I began taking thyroid medication after my diagnosis of hypothyroidism, I began to periodically feel unwell as I did when I had high blood sugar.  I began to test my blood glucose more often and discovered that it was routinely spiking as high as the mid- to high 10 mmol/L (~190 mg/dl) for seemingly no reason. 

Effect of thyroid medication on blood sugar

I was totally puzzled as to why.  I didn’t eat simple carbs or starch-based food. I wasn’t sick, or under any new stress. I was sleeping well, was  properly hydrated, and there was no reason that I could think of that my blood sugar would keep spiking. In my search for answers, I stumbled across information that indicated that it should be well known that thyroid medication can worsen blood sugar control in people diagnosed with diabetes. For some reason, this information was not communicated to me, and when I asked others with both disorders, they were also unaware.

While I have already been eating low carb for the past three years, I began eating very low carb in order to get a better handle on the blood glucose spikes, and it is helping. I am also, in conjunction with my doctor, adjusting the timing of my thyroid medication around the timing of my meals to minimize the impact of the thyroid medication on my blood sugar and will continue to monitor my blood sugar several times per day. If need be, I will have my doctor either prescribe a medication such as Metformin to support normal blood sugar and/or trial different doses of thyroid medications. The important factor is I now know and can monitor this and make changes, as necessary. 

How many people have no idea?

Diabetes and Thyroid Medication 

It is essential that people diagnosed with any form of diabetes (type 1, type 2, gestational diabetes) as well as hypothyroidism know that their thyroid medication can impact their blood sugar control, as well as their need for diabetes medications, if they take any. Since those with type 1 diabetes and gestational diabetes have to monitor their blood glucose very closely, they would notice any changes, but many people with type 2 diabetes rarely regularly check. 

Thyroid Medication — highlights of prescribing information (product monographs) 

The “Highlights of Prescribing Information” sheets for both Synthroid® and Cytomel® that are available to doctors and pharmacists warn that therapeutic used of “these medications in patients with diabetes mellitus may worsen glycemic control and result in increased antidiabetic agent [i.e., diabetes medications] or an “increase in insulin requirements.” In addition, there is a warning on prescribing both medications to “carefully monitor glycemic control (i.e., blood sugar) after starting, changing, or discontinuing thyroid hormone therapy.” This means that people with diabetes need to be continuing to monitor their own blood sugar and contact their doctors if there is a need to address worsening blood sugar control related to taking thyroid replacement medication.

The “Highlights of Prescribing Information” for Synthroid® [1] is a 19-page product monograph about this medication which states that it does “not include all the information needed to use this medication safely and effectively.” For that, there is the need to read the full prescribing information. In other words, these 19 pages are only a summary of all that is needed to use the medication safely and effectively! 

Below is page 1 of the 19 of the “Highlights of Prescribing Information” for Synthroid®.

Highlights of Prescribing Information for Synthroid – page 1 of 19 highlighted

If the relevant section wasn’t highlighted in yellow by me, how likely would it be that a pharmacist or physician would have noticed this warning amongst the 19 pages of fine type?

The “Highlights of Prescribing Information” for Cytomel® [2] is an 11-page product monograph about that medication. It also states that it does “not include all the information needed to use this medication safely and effectively.” Those sheets are just a summary, and there is the need to read the full prescribing information.

Below is page 1 of the “Highlights of Prescribing Information” for Cytomel®.

Highlights of Prescribing Information for Cytomel – page 1 of 11 highlighted

Again, if the relevant section wasn’t highlighted in yellow by me, how likely would it be that a pharmacist or physician would have noticed this warning amongst the 11 pages of fine type?

 


[Post publication note (May 1, 2023)] 

Since natural desiccated thyroid (NDT) also called natural desiccated extract (NDE) are not approved by the FDA or Health Canada as medications, there are no Prescribing Information sheets for products such as the US product Armour Thyroid®, or equivalent ERFA desiccated thyroid®,  in Canada, but both products contain the same warning.

Page 3 of 24 of the Product Monograph from Armour®  contains a warning under Contraindications;

“Thyroid hormone therapy in patients with concomitant diabetes mellitus or diabetes insipidus or adrenal cortical insufficiency aggravates the intensity of their symptoms. Appropriate adjustments of the various therapeutic measures directed at these concomitant endocrine diseases are required.”

Page 3 of 24 of the Product Monograph from Armour thyroid®

Page 1 of 9 of the Product Monograph from ERFA desiccated thyroid®  contains the same warning under Precautions;

page 4 of 9 Product Monograph Erfa Thyroid®

Recently published studies report that 11%–23% of people with type 2 diabetes also have hypothyroidism [6] making it essential that people with both diagnoses know about the possible effect of thyroid replacement on blood sugar control.

For those interested in the mechanism, a paper published last month explains how thyroid hormones contribute to a rise in blood glucose.  In the liver, thyroid hormones increases expression of glucose transporter 2 (GLUT2), which increases in both gluconeogenesis and glycogenolysis.  In liver, the thyroid hormone T3 increases gluconeogenesis by increasing activity of phosphoenolpyruvate carboxykinase (PEPCK), and in adipose tissue, thyroid hormones increase lipolysis, resulting in an increase in free fatty acid that stimulates hepatic gluconeogenesis [7].

Additional References

4.  Product monograph for Armour Thyroid: https://dailymed.nlm.nih.gov/dailymed/getFile.cfm?setid=56b41079-60db-4256-9695-202b3a65d13d&type=pdf

5. Product monograph for Erfa: https://pdf.hres.ca/dpd_pm/00034857.PDF

6. Talwalkar P, Deshmukh V, Bhole M. Prevalence of hypothyroidism in patients with type 2 diabetes mellitus and hypertension in India: a cross-sectional observational study. Diabetes Metab Syndr Obes. 2019 Mar 20;12:369-376. doi: 10.2147/DMSO.S181470. PMID: 30936734; PMCID: PMC6431000.

7. Eom YS, Wilson JR, Bernet VJ. Links between Thyroid Disorders and Glucose Homeostasis. Diabetes Metab J. 2022 Mar;46(2):239-256. doi: 10.4093/dmj.2022.0013. Epub 2022 Mar 24. PMID: 35385635; PMCID: PMC8987680.


Final Thoughts…

If you have any type of diabetes and have also been diagnosed with hypothyroidism (including Hashimoto’s disease, the autoimmune form), it is very important that you monitor your blood glucose regularly. 

Contact your doctor if you notice a worsening in your blood sugar control, and to have your doctor evaluate your need for an increased dosage of existing diabetes medications, or the introduction of these medications if you don’t currently take any.

Consider adopting a style of eating that is lower in carbohydrate than you currently eat. According to a 2019 consensus report from the American Diabetes Association, reducing overall carb intake has “the most evidence for improving glycemia [blood sugar]” [3] . But please keep in mind that while a low carb diet is safe and effective for those with diabetes, if you take certain types of medications it is necessary to have medical oversight before adopting a very low carbohydrate (“keto”) diet.

More Info

If you have diabetes or pre-diabetes* and would like information on how I can support you in better managing your blood sugar, please send me a note through the Contact Me form at the top of this page.

To your good health!

Joy

*Please note that I do not design Meal Plans for people currently taking insulin or insulin-analogue medication for diabetes as I do not have CDE certification.

You can follow me on:

Twitter: https://twitter.com/JoyKiddie
Facebook: https://www.facebook.com/BetterByDesignNutrition/

 

References

  1. Food and Drug Administration, Highlights of Prescribing Information for Synthroid, https://www.accessdata.fda.gov/drugsatfda_docs/label/2020/021402s034lbl.pdf
  2. Food and Drug Administration, Highlights of Prescribing Information for Cytomel, https://www.accessdata.fda.gov/drugsatfda_docs/label/2018/010379s054lbl.pdf
  3. Evert, AB, Dennison M, Gardner CD, et al, Nutrition Therapy for Adults With
    Diabetes or Prediabetes: A Consensus Report, Diabetes Care, Ahead of Print, published online April 18, 2019, https://doi.org/10.2337/dci19-0014

 

Copyright ©2023 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

 

Hypothyroidism Update – six months of treatment since diagnosis

This update marks just over six months since I was diagnosed with profound hypothyroidism and began medication and nutritional treatment. It is written from a subjective perspective and thus is categorized as part of my personal story.


At the beginning of June 2022, our family was in Tofino (Vancouver Island) for the marriage of my youngest son. The groom’s eldest brother assumed that my inability to walk on the sand for family photos, up the path to the hotel, or to get up out of a chair was a result of me having “aged.” He had no idea that I was hiking in North Vancouver and Golden Ears Provincial Park for several hours at a time just the summer prior.

I knew something was wrong, and for several months, I assumed my feeling exhausted and having joint and muscle pain was a carry-over effect from having had Covid. But a cell phone picture of myself taken just before the wedding told me it had to be something else. Gradually, over several months, I went from looking as I had been the previous two years after losing 55 pounds to looking like I had regained everything. I later found out, it wasn’t fat but an accumulation of mucin in the skin that is one of the hallmark signs of myxedema. You can read more about myxedema and the skin changes associated with hypothyroidism here

Since it was a special occasion, I didn’t say anything to my family about how sick I felt, but I was beginning to think that I had become significantly hypothyroid since I last saw my doctor in person (due to Covid protocols). My plan was to contact him when I returned to the mainland, which I did.

Two weeks later, my doctor confirmed that my symptoms were consistent with a diagnosis of hypothyroidism. In fact, I was surprised when he mentioned that it was not unexpected in light of my lab work over the previous nine years, my past thyroid surgery several decades ago, and my having experienced periodic hypothyroid symptoms since that time. Nevertheless, it took almost a decade for me to get diagnosed because of the limitations placed on doctors regarding which tests they can requisition under what circumstances (more about the challenges of getting diagnosed with hypothyroidism here).

In addition to the clinical challenges of getting diagnosed, there is also the reality that the most common symptoms of hypothyroidism are often assumed to be “just aging.” For example, many people believe it is normal for ‘older adults’ to have body aches, joint pain, fatigue, feel chilled when others do not, experience constipation, have dry skin or hair loss, be forgetful, or to even experience depression. Unfortunately, many don’t realize that these are not typical signs of aging but ARE common symptoms of hypothyroidism. What compounds the challenge of getting diagnosed is that the symptoms of hypothyroidism are so non-specific that many would not give them a second thought. An older person limited to a “one-issue-ten-minute remote doctor’s appointment” would be unlikely even to bring them up.

For those who have been following this story, my diagnosis was not the end but the beginning of my journey. Three months later, I lost half my hair due to telogen effluvium, the most common form of diffuse hair loss that can occur after a profound stress, shock or traumatic event including childbirth, a thyroid disorder, or rapid weight loss. You can read more about that here.

When my hair loss continued due to androgenic alopecia (also common in hypothyroidism), I began to research which nutrients of importance had evidence for helping restore hair loss, and wrote this article. Knowing I had a second son’s wedding mid-February, I incorporated both nutritional supplements (oral and topical) to support me in my recovery from what my doctor called “profound hypothyroidism.”

This weekend was my second son’s wedding, and the difference between how I felt in June and now is incredible! Instead of wearing medical compression stockings and orthopedic shoes so I could walk, I wore regular nylons and dress pumps.

While my doctor said it would still take another six months or longer for the mucin to resolve in my legs and trunk of my body, I was SO pleased that my legs didn’t look like water-logged tree stumps, as they did in June! In addition, my face was no longer swollen beyond recognition. I looked like “me” rather than like I had been “inflated” with an air pump.  I felt human and presentable and unlike I did in June, I wasn’t self-conscious being in the family photos.

Looking at the two wedding pictures side-by-side (see below), it is evident that being on the correct dose and mix of thyroid hormones (thanks to the excellent support of my doctor) has made a significant difference! In addition to thyroid medication, I have also been supplementing with nutrients of importance in hypothyroidism, as well as nutritional supplements with evidence to restore hair loss in androgenic alopecia which I developed secondary to my diagnosis. My hair is gradually growing back in, and where once there was a bald shiny scalp, I have hair an inch or two long. I also have eyelashes again, and the outer thirds of my eyebrows are also coming back in. 

 

It is my hope that when the most recent newlyweds celebrate their first anniversary, that the residual symptoms of hypothyroidism will be behind me.

Final thoughts…

If you have wondered if you have symptoms that may be consistent with hypothyroidism, you can download a checklist of common hypothyroid symptoms here to help you have an informed discussion with your doctor to determine whether thyroid hormone testing is warranted.

More Info?

If you would like more information about how I could support you from a nutritional perspective, please send me a note through the Contact Me form at the top of this page.

To your good health!

Joy

You can follow me on:

Twitter: https://twitter.com/JoyKiddie
Facebook: https://www.facebook.com/BetterByDesignNutrition/

 

Copyright ©2023 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

 

 

Alopecia Universalis – a clinician’s personal story of complete hair loss

Three years ago, Tim Rees, a clinician from Germany, lost all his hair a second time to alopecia universalis (AU).  As a registered clinical nutritionist he felt he had lost his credibility to help others with autoimmune conditions, and when he expressed those feelings in a recent blog (link below), it struck a chord with me. 

Tim Rees with his son in 2022

“All my hair fell out, but it was my response to it that destroyed me. You could be forgiven for thinking this is all centered around vanity, but the thing that crushed me was that it made me feel like a fraud.” ~Tim Rees, registered clinical nutritionist

As a clinician, I felt similarly when I lost half my hair in September of 2022. I wondered if others would consider me a “failure” for not having been able to prevent it.

Neither Tim nor I are physicians and thus are not qualified to diagnose conditions (in ourselves or in others). Our role is to provide nutrition education or medical nutrition therapy for conditions diagnosed by physicians. 

I was so struck by Tim’s recent post sharing about his complete hair loss and how he felt about it as a clinician, that I asked his permission to share his story. Below is an excerpt of his recent post. My goal in sharing this is so that people can understand what alopecia universalis is, and how it feels as a clinician to be diagnosed with an auto-immune disorder. It is my hope that sharing Tim’s post will enable people to better understand that clinicians with health conditions (whether autoimmune or not) are no less able to support their clients. What makes a clinician knowledgeable is their training and ongoing study in their area of clinical practice and I do not believe that a clinician diagnosed with an autoimmune disorder or metabolic disease is disqualified from being able to help others. On the contrary, provided they remain objective, I think a clinicians’ ability to understand their clients’ clinical struggles from “both sides of the clinical desk” while offering evidence-based support may be an asset.

I will begin with a very brief explanation of the disorder itself, so Tim’s words make sense.

Alopecia Universalis

Alopecia universalis (AU) is an advanced form of alopecia areata (AA) which is a condition that causes round patches of hair loss. This recent article describes alopecia areata and shows pictures of what it looks like.

In alopecia universalis, there is a complete loss of hair on the scalp and all over the body and it is thought to be an autoimmune condition in which the person’s immune system mistakenly attacks the hair follicles [1].


This Year I Stopped Hiding – a clinician story

(written by registered clinical nutritionist, Tim Rees)

“Three years ago my hair started falling out for the second time. In fact, I’d only had it all back for about 6 months before I got gut-punched standing before the mirror. “It’s not as bad as last time,” I said to myself.

But like an unstoppable rebel force (name the movie) my immune system killed my hair follicles and the hair dropped away like oak leaves in autumn, minus the orange. Alopecia Universalis, not a single hair remained on or in (I’m told) my body.

At the same time, the entire world went into lockdown and the corporate presenting side of my business died along with my self-esteem, my confidence and my monthly hairdressing appointment. There had never been a better time to hide.

You could be forgiven for thinking this is all centered around vanity, but the thing that crushed me was that it made me feel like a fraud. I was so embarrassed the thought of people discovering my secret presented as physical pain. Alopecia, one of the most visible autoimmune diseases one can have, undermined my work and, I thought, my credibility as a nutritionist helping people with autoimmune conditions.

But that’s not true. I’ve done amazing things with nutrition for myself and my clients. Until fairly recently I had lost my hearing to the point that I could no longer use the phone and was conducting sessions using Skype subtitles and talking non-stop in the hope I’d cover their questions before they thought them up. I have a whole list of reasons-why-I’m-not-a-fraud but I won’t bore you with them, after all, most of these insecurities are in my head.

But, here’s the thing. I think I can reverse this condition. Two years ago I stuck to my exclusion diet for four months by which time I had quite a lot of regrowth. Fluffy like a baby owl but still, living follicles. In fact, I remarked to my helpless doctor that I wasn’t worried about the hair, that it was coming back and all was fine. Ever the optimist. But after some bad luck, I began compromising a little here, and a little there, it was Christmas after all, and before I knew it I was doing a passable impression of a bowling ball again.

This year will be different. I’m plastering this all over social media for a number of reasons. Firstly, it’s a part of my acceptance. I fought hard against acceptance mistaking it for defeat. The truth is, you must accept how things are today in order to make a difference tomorrow.

Secondly, after posting a couple of photos on Twitter, I already feel better. And, thirdly it’ll help to keep me motivated and compliant for however long it takes to allow my body to heal.

I’ve been drifting and failing as a husband, as a new father and as a man. Not because I have alopecia but because I’ve let it destroy me. There’s a stoic lesson in there.”

[Shared with permission from Tim Rees’ blog.]


As Tim outlines in the full article, it is his goal this year to reverse his alopecia universalis, and like I did when I set out to recover my own hair loss from telogen effluvium and androgenic alopecia, he will be sharing his progress on social media for all the world to see. 

Tim plans to use an exclusion diet as well as nutritional supplements and to document why he is using them.  He also intends to integrate other approaches which he hopes will support his goal, including the use of sauna, cold thermogenesis, exercise, circadian rhythm / light exposure, etc. and document what he found helpful. While this will be Tim’s personal account of what he is doing to improve his hair loss, I am confident that as a clinician, he will document his choice of approaches and provide references.

I applaud Tim’s boldness and bravery to stop “hiding” and to live his hair loss story and goal of hair loss restoration in a public way. I wish him all the very best in achieving his goal.  

Final Thoughts…

It is important to keep in mind that what may work for Tim may not work for others diagnosed with the alopecia universalis, anymore than the nutrients I took would work for others diagnosed with telogen effluvium and androgenic alopecia. I chose to not write about which nutrients I took and in what dosages because it was not relevant to anyone other than me. I did write two referenced articles related to nutrient supplementation and hair loss and the first one was Hair Loss in Hypothyroidism (Part 2) – Nutrients of Importance  and the second was Nutritional Supplements With Evidence to Restore Hair Loss.

Since taking some nutritional supplements is not without risk, I would encourage anyone considering doing this to first consult with a qualified healthcare professional. Let them assess you to help determine which nutrients may be low or deficient based on dietary intake, and lab work.

A registered clinical nutritionist such as Tim Rees, BSc mBANT rCNHC from Ebersberg, Germany is licensed to support people in that country and I can support people in several provinces in Canada. If you would like more information on how I can help, please send me a note through the Contact Me form at the top of this page and you can reach out to Tim on his blog.

[Please note that I do not know Tim personally and as such this article is not an endorsement.]

To your good health!

Joy

You can follow me on:

Twitter: https://twitter.com/JoyKiddie
Facebook: https://www.facebook.com/BetterByDesignNutrition/

References

  1. National Institute of Health, Genetic and Rare Diseases Information Centre, Alopecia universalis, https://rarediseases.info.nih.gov/diseases/614/alopecia-universalis

 

Copyright ©2023 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

Hair Loss in Hypothyroidism – nutrients of importance

In the previous article titled Hair Loss – root causes was about the three most common types of hair loss, including androgenic alopecia, alopecia areata, and the most common form of diffuse hair loss, telogen effluvium which is the type often associated with hypothyroidism.  This article explains the role of specific vitamin and mineral deficiencies in hair loss and how treating them can help restore hair growth.

As explained in the previous article, telogen effluvium (TE) is the most common form of hair loss in hypothyroidism and is where the hair often comes out in clumps in the shower or a brush. Hair loss is usually from all over the scalp but may occur more on the temples, the part, and the crown of the head [1]. But TE is not the only type of hair loss in hypothyroidism. In a study of more than 1200 people with thyroid disorder, half (50%) of people aged 40 years old and older had either alopecia areata, or androgenetic alopecia [2].

What makes the hair loss associated with thyroid dysfunction particularly challenging is that it occurs 2-3 months after the overt symptoms of thyroid disorder began, which is usually once they’ve already begun thyroid hormone treatment for hypothyroidism.

The pictures below are of me. The one on the left was taken at one of my son’s wedding in June 2022, just prior to being diagnosed with hypothyroidism.  I clearly had the symptom of edema (facial swelling, leg and hand swelling) associated with undiagnosed / untreated hypothyroidism, the hair on my head was minimally affected. The photo on the right was taken three months later, after beginning hormone replacement treatment for hypothyroidism, and the hair loss and shiny scalp is very apparent. 

No hair loss at height of untreated hypothyroidism, telogen effluvium hair loss 3 months later

Hair loss in hypothyroidism

Normally,  90-95% of hair follicles are in the growth (anagen) phase, with only 5–10% being in the resting (telogen) phase. Only a few follicles are in the transitional (catagen) phase [1] at any one time.  At the end of the telogen phase, the hair falls out and under normal circumstances that would amount to ~ 100-150 hairs per day.

Hair growth stages

 

In telogen effluvium, the growth (anagen) phase slows down and up to 50% of the follicles move into the telogen phase, where shedding occurs. i.e., hair loss becomes 5-10 greater than normal, with people losing up to 50% of their hair.  As can be seen in the photo above, at 3 months I had lost 50% of my hair. 

It wasn’t only half the hair on my head that I lost, also lost 1/2 my eyelashes and part of the outer third of my eyebrows.

Since the period of the most dramatic loss occurs approximately 2-3 months after the triggering event, many people don’t relate the shedding to the event that caused it. 

Hypothyroidism can result in hair loss, but nutrient deficiencies can sometimes underlie hypothyroidism (such as in iodine or iron deficiency) and can often make the symptoms of hypothyroidism worse.  Each person is different and the degree to which underlying nutrient deficiencies may make hair loss worse, varies.  As a result, the sufficiency of the key nutrients related to hypothyroidism should be evaluated. 

If any of these nutrients are found to be deficient or suboptimal, correct supplementation can support the regrowth of hair, but it should be noted that the timing of supplements with respect to each other and in relation to the timing of thyroid medication is essential. The reason for this is that some nutrients complete for binding sites (e.g., iron, zinc and copper) and need to be taken separated from each other. In addition, thyroid medication needs to be taken at least a half hour before and food or vitamin / mineral supplementation, or two hours afterwards. When there are several nutrient deficiencies and multiple doses per day of thyroid medication, this can take quite a bit of planning to get the timing right.

Iron deficiency is very common and one of the deficiencies that contributes to telogen effluvium [3,4], and iron is often low in hypothyroidism [5]. In some cases, treating iron deficiency may in itself be sufficient to restore thyroid function [5]. The reason is that the body requires sufficient iron to convert the inactive thyroid hormone thyroxine (T4) into the active thyroid hormone triiodothyronine (T3) and insufficient iron stores could interfere with this conversion. 

It has been recommend that to reverse significant hair loss due to telogen effluvium to maintain serum ferritin at levels of >157 pmol/L (70 ng/dL) [4].

Some of the best food sources of heme iron (the most bioavailable form) are oysters, clams and liver.

Adequate vitamin C intake is required for intestinal absorption of iron, so ensuring adequate vitamin C intake is important those with hair loss associated with iron deficiency.

Selenium was identified in the 1990s as a component of the enzyme that activates thyroid hormone through the conversion of (inactive) T4 to (active)T3 [6]. Selenium is also used to by the body for the formation of glutathione, a powerful antioxidant that protects the thyroid from inflammation and oxidative stress.

Food sources of selenium include Brazil nuts, with 2 Brazil nuts meeting the daily requirement of 200 mcg of selenium. Other good sources of selenium are mushrooms, eggs, fish such as cod and halibut, chicken and eggs. 

Selenium deficiency is a significant problem in the developing world, but thought to be rare in the West. Research from 2012 indicates that the selenium content of the soil in the US was already lowest in the major agricultural areas of the Northwest, Northeast, Southeast, and areas of the Midwest near the Great Lakes[7] and at the time, only the Great Plains and the Southwest were reported to have adequate selenium content in the soil [6].

Zinc plays a key role in the metabolism of thyroid hormones, specifically by regulating the enzymes that are involved in the activation of T4 to T3, as well as regulating thyrotropin releasing hormone (TRH), and thyroid stimulating hormone (TSH) synthesis [8]. Zinc also modulates structures of essential transcription factors that are involved in the synthesis of thyroid hormones, as well as influence the levels of TSH, T4,  and T3 in the blood [8]. It is important to be tested first to know if there is a zinc deficiency before taking a supplement, because supplemental zinc can result in a reduction in copper, and if taking zinc, it is important not to take it with iron or calcium supplements as they complete for binding sites.

Eating foods rich is zinc is the safest way to ensure adequate intake and good sources of zinc include red meat, poultry, seafood such as oysters, crab and lobster, as well a nuts. 

Vitamin D – in Canada which is above the 49th parallel, it is  known that between 70% and 97% of the population demonstrates vitamin D insufficiency, with 32% in Canada being Vitamin D deficient [9].  Deficiency of Vitamin D in the US is even higher, at 42% [10]. It has been known that there was a relationship between Hashimoto’s (autoimmune) hypothyroidism and Vitamin D deficiency [11], it is now known that non-autoimmune hypothyroidism is associated with vitamin D deficiency [12]. A randomized, double-blind, placebo-controlled trial from 2018 in over 200 hypothyroid patients aged 20-60 years old found that supplementing with vitamin D improved TSH levels and calcium levels in hypothyroid patients [13]. 

In addition to dairy foods that are fortified with Vitamin D, foods that are naturally good sources of Vitamin D include fatty fish such as salmon, mackerel and tuna.

 

Vitamin B12  – It is known that people with Hashimoto’s disease (autoimmune hypothyroidism) have a higher prevalence of pernicious anemia [14], which is caused by a deficiency of vitamin B12, either due to a lack of B12 the diet or an inability to absorb it. In addition, vitamin B12 deficiency can mimic many of the symptoms of hypothyroidism such as fatigue, weakness, yellowish skin, some of the mental health symptoms. The best sources of vitamin B12 are organ meats, including liver and kidney, clams, sardines, and beef.


[UPDATE: December 11, 2022] The photo on the top, below was taken three months after being diagnosed with hypothyroidism and beginning hormone replacement treatment. The hair loss is obvious, as is my shiny scalp. The photo on the bottom was taken today — three months later. It clearly shows the regrowth of hair which is the result of both hormone replacement treatment, and three months of nutrient supplementation to support regrowth. [Note: Each person’s results will be different of course, depending which nutrient deficiencies they may have, and whether these deficiencies were due to the hypothyroidism itself, the result of inadequate dietary intake, or both].

 

Hair regrowth after 3 months thyroid treatment and nutrient supplementation

…and the hair regrowth wasn’t only on my scalp.  When I first lost so much hair, I also lost most about half of my eyelashes, too.  A month ago (Nov. 18, 2022), I took a picture of them growing back in, and below is that photo and what they look like almost a month later (December 13, 2022), without any mascara or eyeliner.

Eyelashes growing back in

POSTSCRIPT (November 18, 2022):  In writing this post yesterday, I came across several research papers that referred to the role of several of the nutrients of importance to hair loss in hypothyroidism, to premature hair greying. While my grey hair certainly was not “premature,”  look what I found today! 

A recent study mapped hundreds of proteins inside of hair and found that white hairs contained more proteins linked to mitochondria and energy use which suggests that metabolism and mitochondria may play a role in hair greying. Since thyroid hormones are known to be the major controllers of metabolic rate, it makes sense that hair that was previously dark might turn grey as the result of hypothyroidism, and revert back to dark with thyroid hormone correction. 

[Rosenberg AM, Rausser S, Ren J, et al. Quantitative mapping of human hair greying and reversal in relation to life stress. Elife. 2021;10:e67437. Published 2021 Jun 22. doi:10.7554/eLife.67437]


Final Thoughts…

While treating hypothyroidism is a medical prescription of thyroid replacement medication in an optimal dosage, determining if any nutritional deficiencies may be contributing to the condition, or mimicking its symptoms, is essential.

Having dietary intake assessed and, if indicated, having blood tests to determine if nutrient deficiencies exist and correcting them can go a long way to helping people feel better and supporting regrowth from hair loss.

It is important to remember that taking supplements needs to be done wisely. “More is not better” when it comes to taking nutrient supplements.

For example, nutrients such as selenium can be toxic in excess amounts, even when eaten as Brazil nuts.

Some nutrients, such as biotin which is often taken by people for hair growth can interfere with thyroid hormone tests.

Iodine is another nutrient that should not be supplemented when people are taking thyroid hormone replacement medication.

If you aren’t sure if your nutrient intake or nutrient status of specific nutrients sufficient, then having a nutritional assessment and blood tests when needed is a great place to start. 

More Info?

If you have been diagnosed with hypothyroidism and would like to better understand the condition and make sure that you have adequate intake of nutrients known to be important in thyroid health, please send me a note through the Contact Me form.

To your good health!

Joy

 

You can follow me on:

Twitter: https://twitter.com/JoyKiddie
Facebook: https://www.facebook.com/BetterByDesignNutrition/

 

References

    1. Malkud S. Telogen Effluvium: A Review. J Clin Diagn Res. 2015;9(9):WE01-WE3. doi:10.7860/JCDR/2015/15219.6492
    2. Vincent M, Yogiraj K. A descriptive study of alopecia patterns and their relation to thyroid dysfunction. Int J Trichol 2013;5:57-60
    3. Almohanna HM, Ahmed AA, Tsatalis JP, Tosti A. The Role of Vitamins and Minerals in Hair Loss: A Review. Dermatol Ther (Heidelb). 2019;9(1):51-70. doi:10.1007/s13555-018-0278-6
    4. Trost LB, Bergfeld WF, Calogeras E. The diagnosis and treatment of iron deficiency and its potential relationship to hair loss. J Am Acad Dermatol. 2006;54(5):824–844.
    5. Ghiya R, Ahmad S. SUN-591 Severe Iron-Deficiency Anemia Leading to Hypothyroidism. J Endocr Soc. 2019 Apr 30;3(Suppl 1):SUN-591. doi: 10.1210/js.2019-SUN-591. PMCID: PMC6552785.
    6. Winther, K.H., Rayman, M.P., Bonnema, S.J. et al. Selenium in thyroid disorders — essential knowledge for clinicians. Nat Rev Endocrinol 16, 165–176 (2020). https://doi.org/10.1038/s41574-019-0311-
    7. Mistry HD, Broughton Pipkin F, Redman CW, Poston L. Selenium in reproductive health. Am J Obstet Gynecol. 2012 Jan;206(1):21-3
    8. Severo JS, Morais JBS, de Freitas TEC, et al. The Role of Zinc in Thyroid Hormones Metabolism. Int J Vitam Nutr Res. 2019;89(1-2):80-88. doi:10.1024/0300-9831/a00026
    9. Schwalfenberg GK, Genuis SJ, Hiltz MN. Addressing vitamin D deficiency in Canada: a public health innovation whose time has come. Public Health. 2010;124(6):350-359. doi:10.1016/j.puhe.2010.03.00
    10. Forrest KY, Stuhldreher WL. Prevalence and correlates of vitamin D deficiency in US adults. Nutr Res. 2011;31(1):48-54. doi:10.1016/j.nutres.2010.12.001
    11. Botelho IMB, Moura Neto A, Silva CA, Tambascia MA, Alegre SM, Zantut-Wittmann DE. Vitamin D in Hashimoto’s thyroiditis and its relationship with thyroid function and inflammatory status. Endocr J. 2018;65(10):1029-1037. doi:10.1507/endocrj.EJ18-0166
    12. Ahi S, Dehdar MR, Hatami N. Vitamin D deficiency in non-autoimmune hypothyroidism: a case-control study. BMC Endocr Disord. 2020;20(1):41. Published 2020 Mar 20. doi:10.1186/s12902-020-0522-9
    13. Talaei A, Ghorbani F, Asemi Z. The Effects of Vitamin D Supplementation on Thyroid Function in Hypothyroid Patients: A Randomized, Double-blind, Placebo-controlled Trial. Indian J Endocrinol Metab. 2018;22(5):584-588. doi:10.4103/ijem.IJEM_603_17
    14. Ness-Abramof R, Nabriski DA, Braverman LE, et al. Prevalence and evaluation of B12 deficiency in patients with autoimmune thyroid disease. Am J Med Sci. 2006;332(3):119-122. doi:10.1097/00000441-200609000-00004

 

Copyright ©2022 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

 

 

 

To Hell and Back – recovering from hypothyroidism (a Dietitian’s Journey)

Last Monday, I went to the lab for updated blood work, including a thyroid hormone panel, additional thyroid antibody tests, and an iron panel. I have moved past the frustration of there being no pricelist available for consumers who are self-paying for lab tests, and am now focusing on the lab test results, and the dietary changes that I need to make in light of those, as I am recovering from hypothyroidism.


DISCLAIMER: This article is a personal account posted under A Dietitian’s Journey. The information in this post should in no way be taken as a recommendation to self-diagnose, self-interpret diagnostic tests, or self-treat any suspected disorder. It is essential that people who suspect they may have symptoms of any condition consult with their doctor, as only a medical doctor can diagnose and treat.


The results came back late Monday afternoon and I met with my doctor to discuss them on Thursday, The good news is that based on calculation estimates converting the bovine Natural Desiccated Thyroid (NDT) medication that I had been taking, to a mixture of Synthroid® (a synthetic T4 medication) and Cytomel® (a synthetic T3 medication), my free T4 and free T3 are almost perfect. As my doctor said when we met, “between your research and my experience, we got this”.

This is a beautiful picture.

 
TSH, fT4 and fT3 levels on T4/T3 thyroid hormone replacement medication

It shows, as my doctor and I had hoped, that my free T3 (the active thyroid hormone) is in the higher part of the reference range (65.5%); a level considered optimal by endocrinologists and thyroidologists who are well-versed in the use of the combination T4/T3 medications that I have been prescribed.

While my free T4 could be in the higher part of the range on the type of medication that I am taking, it may slightly lower because of the feedback from having sufficient free T3. That’s okay! I am feeling so much better, although it will take another year or more until I am really well again.

As expected, my TSH (Thyroid Stimulating Hormone, the pituitary hormone that tells the body how much thyroid hormone to make) is low because the amount of free T3 is optimal. This is a classic feedback loop where free T3 provides feedback on the pituitary gland, indicating that there isn’t a need to make more thyroid hormone. Think of it like a thermostat.  When the room gets warm enough, there is feedback on the thermostat that no additional heat is required, and it turns it off until the room gets cold again.

Of importance, my TSH is not considered “suppressed” (TSH ≤0.03 mU/L) but “low” (TSH = 0.04-0.4 mU/liter) [1], so there is no increased risk of cardiovascular disease or bone fractures. Those with a “high” TSH (>4.0 mU/liter) — which was the level that I was at before being treated, and those with a “suppressed” TSH (≤0.03 mU/L) both have an increased risk of cardiovascular disease, abnormal heart rhythms and bone fractures. Those with “low” TSH (0.04-0.4 mU/liter) like I have, do not [1,2]. So more good news.

I have been diagnosed with Hashimoto’s disease (also known as Hashimoto’s thyroiditis) which is an autoimmune disease and diagnosis is based both on symptoms of hypothyroidism, along with the presence of thyroperoxidase antibodies (TPO-ab) and thyroglobulin antibodies (TG-ab) in the blood [3].

In many cases of hypothyroidism, it is these antibodies that contribute to the gradual disappearance of thyroid cells and the development of hypothyroidism.  In my case, it was the trauma to the thyroid that resulted from surgery that I had 30 years ago to remove a benign tumour that was the major contributor to the eventual decrease in thyroid function.

Prior to being diagnosed, as you can read about here, I had all the classic symptoms of hypothyroidism, including body aches, joint pain, fatigue, feeling chilled, constipation, dry skin, hair loss, being forgetful, and even feeling depressed.

By the point I realized that these symptoms were not consistent with long-Covid (which is what I initially suspected) or aging (which my sons assumed), I had developed some of the symptoms of severe hypothyroidism [3], including difficulty with speech, significant water retention, and peripheral edema (swelling) of the ankles and face [3]. There are more photos of what I looked like when I was very sick here as well as photos from the beginning part of my recovery.

 

To hell and back – 5 months of recovery from hypothyroidism

 

 

The blood tests confirm that I have both thyroperoxidase antibodies (TPO-Ab) and thyroglobulin antibodies (TG-Ab), which along with my symptoms, confirms my diagnosis of Hashimoto’s disease, but thankfully my blood test results indicate that neither are elevated.

Thyroperoxidase-Ab = 9 (<35 IU/mL)

Thyroglobulin Ab = 14 (<40 IU/mL)

While they are not elevated, they are present. 

Gliadin and Transglutaminase

For many years I avoided gluten containing products because I thought I was gluten intolerant, although not celiac.

A year ago that I stumbled across some novel ingredients and had an idea to create low carb breads to provide dietary options for those with diabetes. My goal was to enable people who would not otherwise consider a low carbohydrate diet to be able to adopt one, for health reasons.   I was mainly thinking of those from bread-centric cultures such as South East Asians (Indian) and Hispanics but in time, I developed many more types of low carb bread. 

I was aware of the connection between high gluten consumption and leaky gut syndrome, but against that I weighed the serious morbidity and mortality linked to uncontrolled diabetes. I had come across many people who would rather stay diabetic, and potentially lose their toes or vision than give up bread and developing these breads seemed like the lesser of two evils. 

Since being diagnosed with hypothyroidism that I had been developing over the previous 9 years (more about that here), I learned that the gliadin fraction of gluten structurally resembles transglutaminase. Transglutaminase is an enzyme that makes chemical bonds in the body, and while present in many organs, there are higher concentrations of transglutaminase in the thyroid.

In leaky gut syndrome, gliadin (and other  substances) result in the gaps in between the cells of the intestinal wall to widen. This results in the immune system of the body reacting to food particles that are inside the intestine, that it normally would not see. It is thought that the immune system reacts to gliadin and creates antibodies to it, seeing it as a foreign invader.  Since gliadin and transglutaminase have very similar structural properties, it is thought that in those with leaky gut syndrome, the immune system begins to attack the transglutaminase in the thyroid, and other tissues, contributing to the development of auto-immune conditions, including hypothyroidism. 

A-1 Beta Casein and Gluten

A few years ago, I had leaky gut syndrome but it resolved with dietary changes, including avoiding gluten and A-1 beta casein dairy (you can read about what A-1 beta casein dairy is here).  Naturally, as I had been working on recipe development for the low carb bread book, I had been eating gluten as I tested them. I also became more liberal in including dairy products from A1-beta casein cows, when I hadn’t used it in years. That started when there was severe flooding last year in Chilliwack last year due to heavy rains after the summer, and that was where my goat milk came from.  Even once the roads were open again and the highways rebuilt, I never really went back to using goat milk, which is naturally A-2 beta casein. In the interest of an abundance of caution, I will go back to using dairy products from A-2 beta casein cows, or from goat or sheep milk (that are naturally A-2). Humans produce A-2 beta casein protein, and using milk from A-2 beta casein animals does not result in an immune response. It is not seen as “foreign.”

From what I’ve read and in discussing it with my doctor, it is likely that my hypothyroidism has been developing over the last 30 years, related to the surgery I had to remove a benign tumour. Further supporting that me becoming hypothyroid has been a long time in the making, I have had high-normal levels of TSH over the last 9 years — which happens to be a time period over which I was avoiding both gluten and A-1 dairy. Given that, I think it’s logical to conclude that my hypothyroidism is primarily related to the destruction of thyroid tissue in the invasive surgery connected to removal of the tumour. Further supporting this hypothesis, I currently have fairly low levels of TPO and TG antibodies, so I suspect they have begun developing fairly recently. Since a 2018 study reported that  both TPO-antibodies and TG antibodies are decreased in hypothyroid patients following a gluten-free diet [4], it seems wise for me to go back to avoiding gluten, with the goal of lowering my TPO-antibodies and TG-antibodies down to as close to zero, as possible.

Cruciferous Vegetables

Cruciferous vegetables such as Brussels sprouts, broccoli, bok choy, cauliflower, cabbage, kale are known goitrogens. Goitrogens are naturally occurring substances that are thought to inhibit thyroid hormone production. The hydrolysis of a substance known as pro-goitrin that is found in cruciferous vegetables produces a substance known as goitrin, that is thought to interfere with thyroid hormone synthesis [5]. Since cooking cruciferous vegetables limits the effect on the thyroid function, and eating cruciferous vegetables have many health benefits, I will usually eat them cooked, but not in huge quantities. There are studies that found a worsening of hypothyroidism when people ate very large quantities of these (e.g. 1 – 1 ½ kg / day) so it is recommended that intake of these vegetables be kept relatively constant day to day, and limited to no more than 1-2 cup / day. I’ve decided that when I do eat them, to keep intake to the lower end of that range, and eat more non-cruciferous vegetables instead.

Iron Deficiency and Low Stomach Acid (hypochlorhydria)

I now know why I am still so tired. I asked my doctor to run an iron panel and the results show I have low iron. Previous results indicate my vitamin B12 are fine and I continue to supplement methylated folate and B12, so I know those are not a problem.

While my iron stores (ferritin) are okay, they are not optimal i.e., ferritin = 93 (15-247 ug/L) instead of >100ug/L.

My hematology panel is low-normal i.e. hemoglobin = 122 (115-155 g/L), hematocrit = 0.37* (0.35-0.45 L/L), MCV = 88 (82-98 fl), MCH = 29.5 (27.5-33.5 pg), MCHC = 334 (300-370 g/L)

My serum iron and iron saturation are very low i.e., serum iron = 11.9 (10.6-33.8 umol/L), iron saturation = 0.15 (0.13-0.50)

Low iron status is common with hypothyroidism, but it was surprising to me because I eat beef liver, or chicken livers every week, and also take a heme polysaccharide supplement (like Feramax®), so it may be due to an absorption problem.

Low stomach acid (hypochlorhydria) is common in hypothyroidism, and since low pH is needed for iron absorption, I have made dietary changes to improve that.

Final Thoughts…

I am very grateful that my doctor recognizes my knowledge as a clinician and is receptive to me advocating for my health. I am incredibly fortunate that he involves me in decisions regarding blood tests, as well as discussing medication types and dosages.  As for the dietary changes and supplementation, he is content to let me handle that!

I hope that out of my experience that I have called “to hell and back” that I am able to help others better understand hypothyroid symptoms, diagnosis and treatment options so that they can discuss them with their doctor.

To your good health,

Joy

You can follow me on:

Twitter: https://twitter.com/lchfRD
Facebook: https://www.facebook.com/BetterByDesignNutrition/

 

Copyright ©2022 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

 

 

Blood Tests and Lab Frustrations – a Dietitian’s Journey

NOTICE: This my personal experience as a private consumer of lab services, and is not related to my profession as a Dietitian. This article is posted in a separate section of the web page titled “A Dietitian’s Journey” which is about my personal health journeys.

“A Dietitian’s Journey – Part I” was about my two year journey recovering from obesity, and poor metabolic health and “A Dietitian’s Journey-Part II” is my current  personal journey recovering from hypothyroidism.

This article is written as a private consumer, which is why it is categorized as a personal account, and an editorial.


This past Monday, I went to the lab to have blood tests to measure my thyroid hormones, anti-thyroid antibodies, and an iron panel. When I had met with my doctor last week, I learned that I would need to pay for the free T3 test because the British Columbia guidelines and protocols for ordering thyroid tests state that a free T3 test is only covered to rule out suspected cases of hyperthyroidism [1]. As I wrote last Thursday, I was “more than willing to pay for a $9.35 test to have all the data.” 

I think most people are aware that the healthcare system is economically stretched, and I certainly understand and accept the need to reduce costs. One way to do that is to restrict the ordering of laboratory tests to only medically justifiable circumstances, which makes good sense. 

While I recognize that I am not objective in this situation, it would seem to me that when someone is on thyroid hormone replacement medication that includes both synthetic T4 and T3 hormones, that the expense of both a free T4 test and free T3 test should be covered by the provincial healthcare system as the cost is justifiable because the prescribing doctor needs to determine if the dosage of both synthetic hormones is adequate, but not too high. 

As I said above, I knew last week that I would be paying for the free T3 test and was fine with that, but what I wasn’t prepared for was that I would be expected to pay three times the cost the government pays for the same test, and that there would no patient-price list available.

When I arrived at the lab on Monday, I was told that the free T3 test would cost $32.00.  I replied that there must be a mistake, because the cost of the test is $9.35. I was informed that the government pays $9.35 for the free T3 test, but the patient-pay cost for the same test is $32.00. I explained to the person at the desk that I could understand the test costing more if there was a set-up fee for a stand-alone test, or for a separate blood draw, but this test was going to be run with others using the same blood draw.  I was informed that $32.00 is the patient-pay cost of the free T3 test regardless of whether it is done with other tests, or by itself.

I asked if I could please see the price list with the patient-pay costs, and was told that there isn’t one. I was asked if I wanted to have the free T3 test period formed, and if I did that I would need to pay $32.00. What choice did I have?  It was not as though I could go to one of the lab’s competitors, as this private lab company is the only one providing laboratory services in this city. 

[NOTE (October 28, 2022: I have spoken to people in other provinces, and it appears from what people have said that the practice of diagnostic laboratories not disclosing patient-pay prices occurs in Manitoba, Ontario, and British Columbia. This practice may also occur in others provinces as well, but I don’t know. This article written as private consumer is about the practice of diagnostic labs not disclosing patient-pay prices to consumers, irrespective of which province the practice occurs in, or by what company.]

I paid the $32.00 for the test because I needed this information to know the effect of the medication on my thyroid hormones, and for my doctor to know whether a medication adjustment was needed. I had the disposable income to pay for it, but what about consumers who need a laboratory test to make health decisions or for their doctor to be able to, and who cannot afford that? 

… and why are patient-pay clients charged 3 times as much as the government pays for the same test?  Even if a private consumer was only requesting a stand-alone test and had to pay the ~$15 blood draw fee, this test would only cost $25, not $32.

After my appointment, I wrote the regional office of the lab company and asked “to have the patient-pay lab prices for British Columbia.” I heard back from a Client Service Advisor who told me that “We do not provide a list of what we charge to patients”.

I was flabbergasted. 

I’ve always made the assumption that private businesses are required to post their prices, or at least make them available when asked.

As an individual consumer, what happened at the lab would be like going to the grocery store to buy food, but none of the items for sale have marked prices. You are required to pick out the things you need, but only find out at the cash register what the price is. 

When you get to the cash, you ask the cashier about the prices, and she tells you there’s no price list,  but she can give you the total cost at the end, and you can either pay, or put the items back. Needing the items, you pay what you are told, and take your receipt.

When you get home, you decide to write the head office and ask if they can send you a price list, and are told there IS one, but that they can’t give it to you.

[UPDATE October 29, 2022: The way things are currently set up, one has to make an appointment with the lab, go there, line up and give the person at the desk their requisition, and only then can find out how much the patient-pay part will cost.

After investing so much time, consumers are put in a position of having to make a decision on the spot — pay whatever is being asked, or leave without the test.

Consumers should be able to access the prices online and make a decision at their leisure, before investing so much time.] 

I don’t know whether private businesses in Canada required to post their prices, or make them available when asked. I’ve always assumed they were, but I could be wrong. If there is a requirement to do so, do diagnostic labs have an exemption that enables them not to make their prices available to members of the public?


UPDATE October 28, 2022: I have since found out the same company provides a price list to allied health professionals so that they can provide laboratory assessment services to their clients, and if they choose they can mark up the cost in their own billing.

There are 2 versions of this test list available. They are identical except the one for British Columbia does not have the prices indicated, whereas the Ontario one does (see below).

I have also since found out that the company DOES have patient-pay price list that is titled “British Columbia Private Price List for Commonly Ordered Lab Tests” and is dated April 2021. It is marked “confidential” and as a result cannot be publicly shared.  See #3, below.

    1. The allied healthcare price list available in Ontario, dated November 2018 has the prices marked. See below.
    2. The allied healthcare price list available in British Columbia, dated June 2020 does not have the prices marked.

Above is the allied health professional cost (November 2018) for an entire thyroid panel of 6 thyroid-related lab tests, including;

          • TSH
          • free T4
          • free T3
          • reverse T3
          • thyroperoxidase antibody (TPO)
          • anti-thyroglobin antibody (TG-ab)

Compared to what the BC government pays for the same tests (minus the reverhttp://from http://www.bccss.org/bcaplm-site/Documents/Programs/laboratory_services_schedule_of_fees.pdfse T3 which isn’t paid for by MSP) the above panel costs $80. Presumably naturopaths are charged prices similar to what MSP pays.

3.    I have since found out that there IS a patient-pay price list and it is titled “British Columbia Private Price List for Commonly Ordered Lab Tests” and is dated April 2021.

 

The prices cannot be posted because the notice at the top of the price list reads;

“This is a confidential document. Please do not disclose our prices publicly except in conversations with your patients.”

Why is the private-pay price of lab tests a confidential document, and why can’t the prices of lab tests be disclosed to the public?

Are business in British Columbia required to disclosed their prices and if so, are diagnostic labs exempt from making their private-pay prices available to consumers?

I don’t know.

How many people would be willing to order dinner at a restaurant that did not post the price of its menu items until after they ordered?

 

My Thoughts on Patient-Pay Prices

I believe that as consumers, private-pay individuals have a right to have access to the prices for laboratory tests in advance, so that they can consider their decision to purchase, or not purchase these services. Consumers expect grocery stores and department stores to post their prices, and it is my personal opinion that privately owned laboratories from whom private consumers purchase services should be no different.

I also think private-pay individuals have a right to know why they are required to pay a premium price for the same services that the government gets for a third the cost, and allied healthcare professionals obtain for approximately half the cost.

This differential pricing for allied health professionals is a little like retailers selling supplements to practitioners at wholesale prices, while expecting the consumer to pay full price. Even car dealerships have “employee pricing” events so that the average consumer can take advantage of the same discounts provided to their employees, but at these diagnostic labs, consumers are unable to know in advance how much they will be paying for services before they arrive at the cash.

I believe that as private businesses, diagnostic laboratories are free to set their prices as they see fit but it would seem that (1) consumers should be able to know what those prices are in advance, and (2) that consumers should also know that they are paying a premium price for the same services, compared to what the government and allied health professionals are paying.

I am very grateful to live in a country where publicly funded medical care is available. I am thankful to have access to excellent diagnostic lab tests, and don’t even mind paying the same cost the government pays for tests that I want to have done. But as a private consumer, I believe the cost of services need to be available and that there needs to be transparency with regards to pricing discounts provided to others.

To your good health,

Joy

You can follow me on:

Twitter: https://twitter.com/lchfRD
Facebook: https://www.facebook.com/BetterByDesignNutrition/

 

References

  1. BC Guidelines & Protocols Advisory Committee, Thyroid Function Testing in the Diagnosis and Monitoring of Thyroid Function Disorder, October 24, 2018

 

Copyright ©2022 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

 

What Does Success Looks Like Now – A Dietitian’s Journey

This article is the fourth entry in A Dietitian’s Journey and is about how I will measure success as I recover from hypothyroidism.

A Dietitian’s Journey – Part I

“A Dietitian’s Journey” (Part I) was my personal weight-loss and health-recovery journey that began on March 5, 2017 when I decided to make dietary and lifestyle changes so that I could reclaim my health. At that time, I was obese, had type 2 diabetes for the previous 8 years, and extremely high blood pressure. 

Two years later, on March 5, 2019, I accomplished all but one of my goals, and the last one I achieved three months later. In all, I lost 55 pounds and more than a foot off my waist, and met the criteria for partial remission of type 2 diabetes, and remission of hypertension (high blood pressure).

To get an idea of what I looked like at the beginning and the end of that journey, there are two short videos on my Two Year Anniversary post that tell the story well.  The first video was taken when I started and it is very apparent how obese I was, and how difficult it was for me to walk and talk at the same time. The second clip was taken when I completed my journey, and the difference is unmistakable.  

A Dietitian’s Journey – recovery from hypothyroidism

Without much difficulty I maintained my health and my weight-loss from March 2019 until August 2020 but then I came down with Covid.  This was at the very beginning of the pandemic and no one really knew what to expect in terms of symptoms. As you can read about in the first post in what has effectively become A Dietitian’s Journey Part II, (When a New Diagnosis is a Long Time Coming ) I had symptoms that both my doctor and I assumed were related to the virus, including muscle aches and joint pain, being exhausted, having ‘brain fog,’ headaches, and having the shivers.

Afterwards, I had to work very hard to regain my mobility. No one knew this wasn’t ‘normal.’

At first, I could barely walk up a flight of stairs. At the time, “success” was being able to walk around the block.  Then I began taking several dietary supplements to help strengthen my immune system and in retrospect, the reason I felt better was likely due to the fact that these were all supplements involved in thyroid support. Success at the time was being able to walk around the man-made lake at the local park, but over the weeks and months of supplementing my diet and walking every weekend, success was being able to complete several medium difficulty hikes in the local mountains. 

Unfortunately, in March of 2022,  I came down with what my doctor assumed was Covid again. At first the symptoms were similar to what I experienced in August 2020, including muscle aches, joint pain, being exhausted, feeling cold all the time, with the only difference being that I didn’t have headaches. The symptoms persisted for several months and I was beginning to think that I had “long-Covid.” As most people did over the pandemic, I put on 20 pounds, but from March to May, I began to look as though I was putting on significant weight, but every time I got on the scale it indicated only a few pounds of difference. I had no idea what was going on.

The next symptom that I became aware of was swelling in my ankles. It wasn’t just a little bit of swelling, but significant enough that I needed to wear compression stockings all day.

At my youngest son’s wedding at the beginning of June, I looked like I did when I was 55 pounds heavier, but I wasn’t.

LEFT: March 5, 2017, RIGHT: June 3, 2022

About three weeks after the wedding, I was diagnosed with hypothyroidism, and started taking desiccated thyroid. At first, I felt significantly better, and within several weeks, the edema in my legs began to subside. 

 

There is still a fair amount of mucin accumulation in my legs, but as of this weekend, I can begin to grab a very small amount of flesh between my fingers. From what I have read it will take at least 6 months for this to resolve. You can read a referenced article about the skin symptoms associated with hypothyroidism here.

It is easy to see from the above photo that in less than 3 months on thyroid medication treatment, my face has lost its puffy, “inflated” look yet amidst the positive improvements of decreased edema and looking more like myself in some respects is the reality that I have lost ~1/2 of my hair due to telogen effluvium that often occurs with sustained hypothyroidism. You can read more different causes for hair loss here.

Loss of half my hair in 3 months due to telogen effluvium.

Even though I have already been on thyroid replacement hormones for several months, it usually takes ~3-6 months for hair loss to stop and another 3-6 months for regrowth to be seen and 12-18 months to complete regrowth [3]. For someone like my who has lost half their hair, six months to a year to begin to see hair growth can seem like an eternity.

I recently changed medication forms from desiccated thyroid to a mixture of T4 medication (Synthroid®) and T3 medication (Cytomel®). The overall distribution of T4:T3 is about the same, but it is hoped that this mixture will result in more stable thyroid hormones day-to-day.

In six weeks I will have new blood tests to re-evaluate whether my levels have improved.  At last check, my TSH was still high-normal (3.47 mU/L) when in most patients on thyroid hormone replacement the goal TSH level is between 0.5 to 2.5 mU/L [7]. My Free T4 =  14.0 pmol/L which is still in the lower end of the range (10.6-19.7 pmol/L) when it is considered optimal to be in the higher end of the range. 

Metabolic Changes due to Hypothyroidism

It’s well known that people with hypothyroidism experience several clinical changes including different type of anemia, changes in how their heart functions, changes in blood pressure, blood sugar and cholesterol and weight gain due to a slower metabolism. My recent medical work up indicates that I was no different in this regard.

Different Types of Anemia

People with hypothyroidism have a decrease in red blood cells and experience different types of anemia, including the anemia of chronic disease. In addition, 10% of people with hypothyroidism develop pernicious anemia, which is associated with vitamin B12 and folate (folic acid). Iron deficient anemia is also common due to decreased stomach acid that results in decreased absorption of iron.

I was supplementing with B12 and folate and as a result have no signs of pernicious anemia, however my hematology panel indicates that I may have iron deficient anemia. An iron panel would be able to quantify this, however I am already taking heme iron supplements, along with vitamin C to support absorption.

Heart Changes

The slowing of metabolism associated with hypothyroidism also results in a decrease in cardiac (heart) output, which results in both slower heart rate and less ability for the heart to pump blood.  This is what results in the unbearable fatigue.

High Blood Pressure

The decreased ability of the heart to pump leads to increased resistance in the blood vessels, which results in increased blood pressure (hypertension).

In those who had normal blood pressure previous to developing hypothyroidism, blood pressure can rise as high as 150/100 mmHg. Hypothyroidism may increase it further for those previously diagnosed with high blood pressure. While my blood pressure had been normal for more than a year, it gradually started increasing the last year, which in retrospect is the period of time over which I was exhibiting more and more symptoms of hypothyroidism. I have since been put back on medication for hypertension to protect my kidneys, which I hope to be able to get off of again within the next six month to a year, as my thyroid hormones normalize.

Weight Gain

Thyroid hormones act on every organ system in the body, but the thyroid is well-known for its role in energy metabolism. When someone has overt hypothyroidism, there is a slowing of metabolic processes, which results in symptoms such as fatigue, cold intolerance, constipation, and weight gain. 

Weight gain is not only about diet or how much someone eats versus how much they burn off. It is also about the person’s metabolic rate, which can be impacted by several things, including decreased thyroid hormones. I gained 20 pounds over the pandemic (much of which overlaps with the period of time over which I was exhibiting more and more symptoms of hypothyroidism. I also gained 10 pounds from March to June which is mostly water weight, due to the mucin accumulation.

High Cholesterol

It has long been known that those with hypothyroidism have high total cholesterol, high low-density lipoproteins (LDL) [4], and high triglycerides (TG) [5], which results from a decrease in the rate of cholesterol metabolism. My doctor deliberately did not want to check these last time, because he knew they would be abnormal only as a result of the hypothyroidism. He plans to evaluate them once I have been stable on hormone replacement for several months.

So, What Does Success Look Like Now?

Just as I had a clear idea of what success looked like in my first A Dietitian’s Journey, I have a clear idea of what I would like success to look like this time, as I recover from my hypothyroid diagnosis.
 

Over the next year, this is what I want to accomplish;

    1. weight same as March 5, 2019 (end of A Dietitian’s Journey, part I)
    2. waist circumference same as March 5, 2019 (end of A Dietitian’s Journey, part I)
    3. regrowth of my hair to same thickness as before clinical symptoms of hypothyroidism
    4. restoration of iron deficient anemia:
      (a) normal ferritin 11-307 ug/L
      (b) iron 10.6-33.8 umol/L
      (c) TIBC 45–81 µmol/L
      (d) transferrin  2.00-4.00 g/L
    5. Blood pressure ≤  130/80 mmHg
    6. Blood sugar:
      (a) non-diabetic range fasting blood glucose ≤  5.5 mmol/L
      (b) non-diabetic range HbA1C ≤  5.9 %
    7. Thyroid Hormones:
      (a) optimal TSH= 0.5 to 2.5 mU/L
      (b) optimal Free T4 = 15-18 pmol/L (10.6-19.7 pmol/L)
    8. Cholesterol:
      (a) LDL ≤ 1.5 mmol/L
      (b) TG ≤ 2.21 mmol/L

Final Thoughts…

While I don’t know if it will be possible to achieve all of these within the time frame or within adjustments to medication that my doctor will be willing to make, these are my goals. I believe that most of these are possible, and as far as they are within my control, this is what I would like to accomplish.

I have achieved a lot the last 3 months, but I am not “done.” I want the rest of my life back!

I want to be able to do the things that I enjoy, and to have the freedom to make plans in the evening knowing I will have the energy to follow through.

I think this is reasonable to ask and I will do everything I can to make this a reality.

A Dietitian’s Journey Part II continues…

To your good health,

Joy

You can follow me on:

Twitter: https://twitter.com/lchfRD
Facebook: https://www.facebook.com/BetterByDesignNutrition/

 

Copyright ©2022 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

 

Hair Loss – root causes (Part 1)

Hair loss can be a very distressing symptom, especially when it is noticeable to ourselves and others. However, before outlining strategies for addressing it, we first need to understand what’s causing it. That is the purpose of this article.  The next article will address strategies for helping to restore hair loss through diet and nutrient supplementation.

There are different types of hair loss with various causes, including genetic, autoimmune, severe stress, as well as nutrient deficiency and nutrient excess. Below are a three of the most common types of loss. 

Male pattern baldness

Androgenic Alopecia is the most common type and affects up to 50% of men and women [1]. In men, it is called ‘male pattern baldness,’ and is mainly seen on the crown of the head and on the temples.  In women, it  is called ‘female pattern baldness,’ and is mainly seen at the crown of the head, with a wider center part [2].  Androgenic alopecia is a genetic disorder that involves both maternal (mother’s) and paternal (father’s) genes, with sons being 5-6 times more likely to have it if their fathers were balding [1]. Since it is genetic, there is no ‘cure,’ but growth may be improved by using products such as minoxidil (Rogaine®) or rosemary extract which has been found to be as effective as minoxidil in studies [2]. One drawback is that treatment needs to continue indefinitely or loss will reoccur when treatment is discontinued [6].

Alopecia areata is an autoimmune disorder where the body’s immune system attacks the follicles. Hair often comes out in clumps, usually the size and shape of a quarter but it can affect wider areas of the scalp [3]. It can occur in those who already have some form of autoimmune conditions, including thyroid disease. Treatment may involve use of oral or topical corticosteroid medication [3] which are very powerful anti-inflammatory medications, or other medications used in autoimmune conditions. Individual bald spots may be treated using Minoxidil (Rogaine®) [3]. 

Telogen effluvium – is the most common form of diffuse hair loss [7]. It usually occurs after a profound stress, shock or traumatic event including after childbirth, as the result of a thyroid disorder, as well as rapid weight loss. It has been reported after a sudden and significant calorie restriction diet (“crash dieting”) [8],  and has also been reported associated with the popularized ‘keto’ diet [9,10], but I am in agreement with Dr. Stephen Phinney of Virta Health that it should not occur in a well-designed keto diet [11].  

In telogen effluvium, hair often comes out in clumps in the shower, or in a brush [6]. Loss is usually from all over the scalp, but may occur more on the temples, the part and the crown of the head [7].  Once the cause telogen effluvium is removed, regrowth will usually begin within two to six months [6].

There are three phases of growth; the growth (anagen) phase, the transition (catagen) phase, and the resting (telogen) phase [5]. During the growth phase, follicles produce a shaft beginning from tip to root [5]. During the catagen and telogen phase, the follicles reset and prepare to start making a new hair. 

hair growth phases – based on Reference [7]

Normal Hair Loss vs Hair Loss in Telogen Effluvium

Normally,  90-95% of follicles are in the growth (anagen) phase, with only 5–10% being in the resting (telogen) phase. Only a few follicles are in the transitional (catagen) phase [7] at any one time.  At the end of the telogen phase, the hair falls out and under normal circumstances that would amount to ~ 100-150 hairs per day [7].

In telogen effluvium, the growth (anagen) phase slows down and up to 50% of the follicles move into the telogen phase, where shedding occurs. i.e., loss becomes 5-10 greater than normal, with people losing up to 50% of their hair.  Since the period of the most dramatic loss occurs approximately 2-3 months after the triggering event, many people don’t relate the shedding to the event that caused it.

Identifying the cause of hair loss is essential, as once identified, and corrected, regrowth will occur [7], but it can take 3-6 months for hair shedding to stop. While many people are anxious that they will go bald, hair loss does not usually exceed 50% of their hair [7].  Once the cause is identified and corrected, regrowth can begin to be seen 3-6 months later [7], but significant regrowth can take 12-18 months [7].

Medications that can interfere with hair regrowth include beta-blockers such as metoprolol and propranolol used in the treatment of abnormal heart rhythms, after a heart attack, or high blood pressure, anti-thyroid medication used in the treatment of hyperthyroidism and anticoagulants [7].

As outlined in this previous article, hair loss is one of the identifying markers of hypothyroidism that results from a lack of thyroid hormones. Hair growth will begin to occur once optimal thyroid hormone replacement is reached, however as mentioned above, it may take 3-6 months for hair shedding to stop, and another 3-6 months for regrowth to be able to be seen [7].  For someone dealing with hair loss, six months to a year to begin to see hair growth can seem like an eternity.  

[I understand this firsthand, as the two photos below are of me.  The one on the left was taken June 3, 2022 at my youngest son’s wedding — a few weeks before being diagnosed of hypothyroidism, and the one on the right was taken yesterday, September 3, 2022, exactly three months later. I share these photos so that people can better understand what the hair loss associated with hypothyroidism may look like.]

Hair loss 3 months after diagnosis

Dr. Izabella Wentz, a clinical pharmacist who focuses on thyroid disorders believes that hair loss is best improved on a medication that contains both T4 and T3, such as desiccated thyroid extract like WP Thyroid®, Nature-Thyroid® or Armour Thyroid®, or a mixture of T4 medication (such as Synthroid®) and a T3 medication such as Cytomel®.  Dr. Wentz also provides a general “rule of thumb” that TSH after treatment should be between 0.5 and 2 μIU/mL [12].

Hair Loss in Nutrient Deficiencies and Nutrient Excess

There are specific nutrient deficiencies that are also linked to different types of hair loss, with the most well-known being iron deficiency. Vitamin C deficiency is also a factor as it is needed  for intestinal absorption of iron.  Zinc deficiency, as well as some B-vitamin deficiency (e.g. niacin, biotin, riboflavin) as well as vitamin D deficiency can also be associated with hair loss [13].  As importantly, excess in vitamins such as vitamin E, vitamin A and folic acid are also associated with hair loss [13]. Ensuring  adequate but not excess nutrient intake is essential and this will be covered in the next part of this article.

Final Thoughts…

Hair loss can be a very distressing symptom, especially when it is noticeable to ourselves and others. Once the cause has been identified and treated, can all we do is be patient and wait for the hair to grow?

Hair regrowth can be supported by ensuring a nutrient-adequate diet, as well as with nutrient supplementation, when there is nutrient deficiency. This will be the topic in Hair Loss – Part 2.

More Info

If you would like more information about how I might be able to support your nutritional needs, please send me a note through the Contact Me form, above.

To your good health!

Joy

You can follow me on:

Twitter: https://twitter.com/JoyKiddie
Facebook: https://www.facebook.com/BetterByDesignNutrition/

 

References

  1. Ho CH, Sood T, Zito PM. Androgenetic Alopecia. Updated Nov 15, 2021. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing, https://www.ncbi.nlm.nih.gov/books/NBK430924/#_NBK430924_pubdet_
  2. Panahi Y, Taghizadeh M, Marzony ET, Sahebkar A. Rosemary oil vs minoxidil 2% for the treatment of androgenetic alopecia: a randomized comparative trial. Skinmed. 2015;13(1):15-21.
  3. Medical News Today, Alopecia areata: Causes, diagnosis and treatments, April 7, 2022, https://www.medicalnewstoday.com/articles/70956
  4. Medical News Today, Is Telogen Effluvium reversible? April 23, 2018, https://www.medicalnewstoday.com/articles/321590
  5. Alonso L, Fuchs E; The Hair Cycle. J Cell Sci 1 February 2006; 119 (3): 391–393. doi: https://doi.org/10.1242/jcs.02793
  6. Phillips TG, Slomiany WP, Allison R. Hair Loss: Common Causes and Treatment. Am Fam Physician. 2017;96(6):371-378.
  7. Malkud S. Telogen Effluvium: A Review. J Clin Diagn Res. 2015;9(9):WE01-WE3. doi:10.7860/JCDR/2015/15219.6492
  8. Goette DK, Odom RB. Alopecia in crash dieters. JAMA. 1976;235(24):2622-2623.
  9. Hallberg, S., Do Ketogenic diets cause hair loss? https://www.youtube.com/watch?v=PxkfM84lxMU
  10. Westman E., Hair Loss and Keto, https://www.youtube.com/watch?v=Cgv92mfTj4k
  11. Phinney S., Virta Health, Does Keto Cause Hair Loss, https://www.virtahealth.com/faq/keto-hair-loss
  12. Wentz I., Hair Loss and Your Thyroid, https://thyroidpharmacist.com/articles/hair-loss-and-thyroid/
  13. Guo EL, Katta R. Diet and hair loss: effects of nutrient deficiency and supplement use. Dermatol Pract Concept. 2017;7(1):1-10. Published 2017 Jan 31. doi:10.5826/dpc.0701a01

 

Copyright ©2022 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

 

Judging By Appearance – a Dietitian’s Journey

We form an opinion about someone’s appearance when we haven’t seen them in a while, or meet them for the first time. We do so unintentionally, but we judge by appearance. Sometimes the appearance of weight gain is not about diet but a diagnosis. 

DISCLAIMER: (August 28, 2022): This article a personal account posted under A Dietitian’s Journey. The information in this post should in no way be taken as a recommendation to self-diagnose, self-interpret diagnostic tests, or self-treat any suspected disorder. It is essential that people who suspect they may have symptoms of any condition consult with their doctor, as only a medical doctor can diagnose and treat.

The photos below are both of me. On the left is what I looked like when I began my personal weight-loss and health-recovery journey on March 5, 2017. Over the following two years, I lost 55 pounds and 12 ½ inches off my waist following a low carb, and then a ketogenic diet. The process was slow — agonizingly slow and in retrospect, I now know why. The photo on the right is what I looked like two years later, maintaining my weight loss.

LEFT: March 5, 2017 RIGHT: December 2021 – after two years weight maintenance

Almost imperceptibly, my appearance began to change.  I didn’t “see it” at the time, but I was aware that my waist circumference was different and that my clothes felt tighter. What I couldn’t understand was that I had only “gained” approximately five pounds.

The two photos below clearly show the subtle difference.

LEFT: Hiking March 5, 2021, RIGHT: Hiking March 5, 2022

The photo on the left was taken on the two-year anniversary of completion of my weight loss journey which lasted from March 5, 2017-March 5, 2019 as posted on my low carb web site. This entry in that journal which is titled From the Mountains Through the Valleys was written for my fifth anniversary, the day before the photo on the right.

The photo on the right was taken this past year in March, exactly one year after the photo on the left.  The comparison is easy because I was wearing the same clothes. While my weight was only approximately five pounds greater than on the left, it is clear to see that my face was puffier, as were my legs.  I remember getting dressed that morning and wondering why all my hiking clothes felt so tight. I also vividly remember how difficult the hike was that day — and it was a simple one with very little elevation. My legs felt heavy, and it was hard to walk up even the gentlest of inclines.

Despite having both vaccines in April 2021 and July 2021, a few days later I came down with what my doctor and I presumed was my second case of Covid-19.

I had Covid the first time in August 2020 and wrote about it in the journey entry titled, To Covid and Back).  In retrospect, I think the ‘post-viral arthritis’ I experienced afterwards may have been linked to my thyroid’s response to the virus (documented in the literature). In that post, I wrote about recovering from Covid the first time;

“By the end of August (after Covid) it was difficult for me to even walk up (or down!) a flight of stairs. This both shocked and scared me.

I began to go for walks — even though it was very hard.  At first they were literally just around the block, but I kept at it.  One of my young adult sons who lives with me kept encouraging me to walk, and would sometimes go with me.  As my legs became stronger, walks turned into short  inadvertent hikes’ and I discovered I really liked being out in the woods, even though it remained very hard to step up onto rocks, or step down from them.  I dug out the wood hiking staff that I brought with me when I moved from California and put it into service., invested in some hiking boots and other essentials’. As I said in the previous article, my hiking stick — along with my fuchsia rain gear has become somewhat of an identifier— but the truth is, without the hiking stick, I could not have possibly begun to hike.

My first breakthrough was in late November, when I did my 4th real hike which was 12 km around Buntzen Lake — which in terms of a few elevation gains was really beyond my capabilities. With frequent stops and lots of encouragement from my son, I did it.  I had to. He couldn’t exactly carry me back to the car! That day I felt as though I had beaten the post Covid muscle weakness and was on my way back to health.”

When I got Covid again this past March, the symptoms were pretty much the same as in August 2020, muscle aches and joint pain, being exhausted, feeling cold all the time and my lips were frequently blue. The only difference was this time I did not have headaches.  I was loaned an oximeter by a family member who is a nurse and I found it quite strange that my body temperature was always two degrees below normal even though I had fever-like symptoms of being cold and shivering.  The muscle aches were significant, as was the fatigue, but since these are also symptoms of Covid, I didn’t think much of it.  It was only when I began to develop symptoms that were not associated with Covid that I began to become concerned.  

Fast forward to the beginning of June which was my youngest son’s wedding. I was so very unwell, but avoided talking about it as I did not want to detract from the very special occasion.

I was experiencing joint pain and muscle aches, and chills that would come and go. I would frequently get bluish lips, and continued to have significant non-pitting edema in my legs and ankles, and was wearing compression stockings all the time — even at the wedding. Most pronounced was the debilitating fatigue.

The skin on my cheeks had become flaky and dry and despite trying multiple types of intense moisturizers, nothing helped. My mouth symptoms had progressed to the point that I found it difficult to say certain words when speaking because my tongue seemed too large for my mouth, and the salivary glands underneath my tongue were swollen.

The muscle weakness had progressed to the point where it was difficult for me to get up from a chair, or to get out of my car without pushing myself up with my hands. My eldest son was helping me get to and from the beach for the photos, and out of the car.  He thought it was me aging, and when I recently asked my other two sons, they assumed the same thing.  I was wondering if I had some form of “long-Covid,” but what got me starting to think that my symptoms had something to do with my thyroid was the very noticeable swelling in my face.

At my son’s wedding I looked like I did when I was 55 pounds heavier!

LEFT: March 5, 2017, RIGHT: June 3, 2022 at my youngest son’s wedding.

The photo on the left, above is what I looked like when I began my weight-lost journey on March 5, 2017. The photo on the right is what I looked like June 3, 2022, at my youngest son’s wedding. I look more or less the same in both pictures, but with a fifty pound difference in weight. 

I found out a few weeks later, I had hypothyroidism and was displaying many of the symptoms of myxedema. [I have written an article from a clinical perspective about the symptoms of hypothyroidism, which is posted here.]

While we do it unintentionally, we all judge by appearance, and “weight gain” is no different. If we see someone at one point in time, we form an opinion based on what we see.  If anyone would have bumped into me three months ago, it would have been reasonable for them to assume that I had gained back all the weight I had lost, and then some. But that wasn’t the case. 

But what causes the appearance of “weight gain,” without gaining significant amounts of weight? 

As I explain in this recent clinical post about hypothyroidism, the “puffiness” is due to the accumulation of mucin under the skin. Mucin is a glycoprotein (a protein with a side chain of carbohydrate known as hyaluronic acid) that is naturally produced in the skin. Under normal circumstances, hyaluronic acid binds water to collagen and traps the water under the skin, keeping it looking moist and plump, In fact, hyaluronic acid is injected into the skin by dermatologists to make aging skin appear younger. The problem in hypothyroidism is that an excess of mucin accumulates under the skin, giving it a “tight, waxy” swollen texture. (I would describe it as feeling like an over-inflated balloon). 

Below is a photo showing the change in appearance in my left leg from November 3, 2021 (left), to July 16, 2022 (middle), to August 26, 2022 (right).

The photo on the left was taken by me last November while I was doing some stretches. It was still on my phone in mid-July when I took a picture of the swelling in my lower legs and ankles caused by mucin accumulating in the skin. The photo on the right was taken this morning, and while much of the swelling has been reduced, I am still unable to pinch any skin on my legs due to the remaining mucin. I have read that it can take 6 – 8 months for this to resolve.

I want people to understand that the appearance of “weight gain” and “weight loss” in hypothyroidism is different than weight gain and weight loss due to dietary changes. The difference, however can be very subtle.

In my case, the appearance of “weight gain” occurred very slowly.

My appearance between March 5, 2021 and exactly a year later are almost indistinguishable. It is only in retrospect, that I can see the puffiness in my face and legs. At the time, I was puzzled why my clothes fit tighter when there was only a 5 pound difference in my weight, but beyond that I didn’t give it any thought.

Below is a composite photo to help illustrate how slowly my appearance changed at first, and how quickly it progressed as my thyroid disorder progressed. Look how rapidly my appearance changed in only three months, between March 5, 2022, and my son’s wedding on June 3, 2022! 

[NOTE: As I’ve mentioned in all of my previous articles and posts about hypothyroidism, each person will present with different symptoms, and even those with the same symptoms may have very different appearance because of differences in their thyroid dysfunction.  Keep in mind, these photos describe only my own experience.]

Below is a composite photo to illustrate how quickly the appearance on my my face has resolved after only two months of thyroid treatment.

An Expanded Perspective

My clinical practice changed 5 years ago when I came to understand what hyperinsulinemia was, and how early clinical signs of developing type 2 diabetes are evident as long as 20 years before diagnosis. In a similar way, my clinical practice is changing again now as the result of what I am learning about hypothyroidism.

Understanding the wide range of clinical and subclinical symptoms that people may have leads me to ask additional questions, to look at lab test results differently, and to ask for additional ones if it seems clinical warranted. While it is beyond the scope of practice of a Dietitian to diagnose any disease or to treat hypothyroidism, I am more aware of what to look and this helps me to refer people back to their doctor if I feel there may be a clinical concern.

Final Thoughts…

We form an opinion about someone’s appearance when we haven’t seen them in a while or when we meet them for the first time. While we do so unintentionally, in developing that opinion, we judge by appearance but sometimes the appearance of “weight gain” is not about diet, but about a diagnosis.

If anyone had seen me three months ago after not seeing me in a while, they might have assumed that I had gained back all the weight I had lost.

When we encounter someone who is overweight, we ought to bear in mind that don’t know where they are on their journey. We don’t know if they have metabolic issues related to glucose and insulin metabolism, are struggling with food addiction, or have an endocrine dysfunction, like hypothyroidism, or something else.

People seeing me now have no idea that less than three months ago I looked as I did on the left, and was very ill.

As much as it is natural for all of us to form an opinion, let’s try not to let that opinion become a judgement.  Listening is a great way to find out more.

To your good health!

Joy

You can follow me on:

Twitter: https://twitter.com/lchfRD
Facebook: https://www.facebook.com/BetterByDesignNutrition/

 

Copyright ©2022 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

More Than Skin Deep – skin symptoms associated with hypothyroidism

According to the American Thyroid Association, 6% of the population have some type of thyroid disease and 60% of them (~12 million people) are unaware of it. Assuming the same rate applies in Canada, 2.3 million people in Canada have thyroid disease and almost 1.4 million people are unaware of it. Since changes in the skin may be one of the first clinical signs of hypothyroidism [2] and are often important indications of its progression [4], this article outlines how some of those skin changes may appear.

DISCLAIMER (August 26, 2022): The information in this post should in no way be taken as a recommendation to self-diagnose, self-interpret skin symptoms or diagnostic tests, or self-treat any suspected disorder. It is essential that people who suspect they may have symptoms of any condition consult with their doctor, as only a medical doctor can diagnose and treat.

NOTE: This article also contains aspects of my personal story which are clearly marked. My personal experience is not objective data. The pictures are provided only so that people can better understand what some skin symptoms of hypothyroidism may look like. Many more pictures are available in clinical online. 


INTRODUCTION: My interest in hypothyroidism is more than academic, as I was recently diagnosed with it. I realize in retrospect that I missed almost all the early signs because I didn’t know what the range of possible symptoms could be. Just as my interest in hyperinsulinemia and type 2 diabetes was birthed in my own diagnosis and eventual partial remission, my interest in this hypothyroidism is no different. Since hypothyroidism can be dangerous if left untreated, my goal in writing this series of articles is to help people know the wide range of symptoms that may be associated with it, and to seek medical attention for themselves or their loved one, when necessary.

As outlined in the article Symptoms of Hypothyroidism Mistakenly Blamed on Aging, people think it is normal for ‘older adults’ to have body aches, joint pain, fatigue, to feel chilled when others do not, experience constipation, hair loss, be forgetful, or to experience depression. However, these are NOT typical signs of aging but ARE common symptoms of hypothyroidism. 

In retrospect, in my case, these types of symptoms came long after the skin symptoms, but cutaneous symptoms were so non-specific that I had no idea they might indicate that something clinical was going on.  

Of course, as a Dietitian, I knew that people often gained weight before they were diagnosed with hypothyroidism, and that they needed to take one of several medications prescribed as treatment. I knew they had to take their medication a half an hour before eating but until recently, my support was limited to teaching them what hypothyroidism is, the nutrients of importance in thyroid function, and foods and beverages that may impact thyroid function.

Until recently, I didn’t know that undiagnosed hypothyroidism can be dangerous and can progress to a myxedema crisis that can be fatal, with a death rate between 20-60%, even with treatment [3]. 

Until today, I had no idea that the majority of people with thyroid disease (60%) are undiagnosed [1].

Putting these two sets of statistics together was concerning to me.  Since many of the symptoms of hypothyroidism such as joint and muscle pain, difficulty getting up from a seated position, or feeling cold are often discounted as normal signs of aging, I wanted people to also know what some of the skin symptoms of hypothyroidism are in the hope that is might help them put the clues together, and seek medical attention.

Skin Symptoms Associated with Hypothyroidism

As mentioned in a previous article about the role of hormones in metabolic disease, thyroid hormones act on every organ system of the body, and their affect on the skin is no exception. Some skin symptoms such as myxedema don’t appear until much later in the progression of hypothyroidism, while other appear early on.

In this article, I will describe the later symptoms first because they are hallmarks of the progression of disease and indicate that getting medical attention is important. In my own case, it was the symptoms associated with myxedema that made me begin to realize that the tiredness and achy muscles and sore joints that I had been experiencing for over a year was more than post-Covid symptoms.

As explained in Symptoms of Hypothyroidism Mistakenly Blamed on Aging, myxedema describes advanced hypothyroidism that occurs when the condition is left untreated or inadequately treated and is also applied to hypothyroidism’s effects on the skin, where it looks puffy and swollen and takes on a waxy consistency [4]. 

[Personal note: It was me looking for clinical answers this morning that resulted in me stumbling across the some of the other skin symptoms associated with hypothyroidism. I wanted to know how long it would take since beginning treatment with thyroid hormone medication for the myxedema to resolve in my legs.]


NOTE: these photos are for illustrative purposes only. Photos of myxedema in the clinical literature are available but are copyrighted. It is for this reason that I am posting my photos only as example, or illustrations.

Below is a photo showing the change in appearance in my left leg from November 3, 2021 (left), to July 16, 2022 (middle), to August 26, 2022 (right).

The photo on the left was taken by me last November while I was doing some stretches. It was still on my phone in mid-July when I took a picture of the swelling in my lower legs and ankles caused by mucin accumulating in the skin. The photo on the right was taken this morning, and while much of the swelling has been reduced, I am still unable to pinch any skin on my legs due to the remaining mucin. I have read that it can take 6 – 8 months for this to resolve.

It has been only 2 months since I began treatment for hypothyroidism, beginning with a very low dose. The above photo shows what I looked like 2 ¾ months ago at my son’s wedding, and how quickly the myxedema in my face resolved with treatment. 


What Causes the Skin Change Known as Myxedema

Myxedema is one several skin significant changes associated with the progression of hypothyroidism. A recently updated dermatology textbook describes myxedema as ‘skin that is cold and pale with abnormally widespread dryness (xerosis) and where a diffuse loss of hair (alopecia) may be present [5].’

When I first saw my doctor after my son’s wedding at the beginning of June, he pointed this out on my legs and said that the cold, waxy skin, along with the swelling is “benchmark symptom” of hypothyroidism.  He showed me how it was impossible to pinch and lift any skin on my legs and that pressing on it left no ‘dent’ mark.  This lack of a dent means the type of edema (swelling) is “non-pitting edema.” Pitting edema occurs in many other conditions, but this non-pitting edema, along with the cold, waxy skin is characteristic of progressing hypothyroidism. The coldness of the skin is the result in the drop in body temperature due to decreased metabolism [2] and is another hallmark symptom of hypothyroidism, discussed in a previous article. The swelling is caused by the accumulation of mucin in the skin.

Mucin is a type of glycoprotein (a protein with a side chain of hyaluronic acid, a sugar molecule) [5] which is naturally produced in the skin. Hyaluronic acid normally binds water to collagen, trapping it in the skin and is injected into the skin by dermatologists to cause aging skin to appear plump, moist and younger looking. The problem is, in hypothyroidism mucin accumulates under the skin, giving it that “tight, waxy” texture. (I would describe it as feeling like an over-inflated balloon). The accumulation of mucin around hair follicles contributes to the resulting hair loss on the arms and legs (and other areas where it occurs). 

[Personal Note: if you look at the composite picture of my left foot (above), you can see in the right hand photo taken this morning (more than 2 months after beginning thyroid hormone medication) that I still cannot pinch any skin on my legs.  While my face has improved, there is still significant improvement yet to occur in my legs, and other parts of my body. ]

Other Skin Symptoms of Hypothyroidism

In addition to myxedema, other skin changes that are associated with hypothyroidism include;

    • dry skin (xerosis)
    • thin scaly skin
    • carotinemia
    • purpura
    • telogen effluvium (hair loss)
    • decrease sweating
    • poor wound healing

As explained in an earlier article, since the presentation of symptoms in hypothyroidism varies so much between individuals, symptoms that were “early” for me, may not be for others, and may not appear at all. 

Purpura is caused when small blood vessels burst, resulting in blood pooling just under the skin. It looks a bit like a bruise, but without pain or swelling and it does not change colour in time.  Purpura is a non-serious skin hemorrhage that is almost always a symptom of something else and looks like small, reddish-purple spots just beneath the skin’s surface.

[Personal account: This morning, when I saw the term “purpura” it jumped out at me. Since May of 2021, I have had a large purple area on my left ankle that I had first attributed to a particularly grueling hike I did in Maple Ridge, BC with one of my young adult sons.  I noticed it when I got home, as did my son, and I assumed it would clear up on its own, but it never did. When I saw my doctor right after my son’s wedding, I showed it to him and he nodded as if to take note, but didn’t say anything. I now know that in my case, it was one of the very early skin signs of hypothyroidism.  I thought I had taken photos of what my purple ankle looked like at its worse, but I may have deleted them because I thought it was simply leftover damage to blood vessels from a hike. The good news is, that two months after beginning thyroid hormone treatment, the purpura is ~75% resolved.] 

August 20, 2022: purpura 75% resolved, thin dry skin, telogen effluvium (hair loss) yet to be resolved

Another early symptom of hypothyroidism for me, was telogen effluvium, a loss of hair on my arms and legs and to a lesser extent, on my scalp. 

[Personal account: Last summer I was joking with a family member that one of the advantages of getting older was no longer needing to shave my legs.  I didn’t realize until recently that the loss of hair on my legs and arms as long as two years ago was NOT a perk of aging (like no longer having a “period”), but was an early symptom of hypothyroidism! I also didn’t realize that decreased sweating wasn’t a benefit of aging, either. I feel stupid in retrospect, but I wasn’t taught it and when I looked it up it said that hair on the body “thins” as one ages, so I thought it was normal.  I hadn’t realized that I had NO hair on my arms and legs. Two months after beginning thyroid medication, that is beginning to change. I feel like a pubescent boy excited by his first facial hair.

I mentioned the dry skin in previous posts, so won’t do so again here, but that was a very early sign for me.  Again, I thought it was a normal part of aging.]

Another term that jumped out at me this morning, was the term carotinemia. This is where beta-carotene accumulates in the blood and gives skin a yellowish pigmentation. In my case, it was not due to eating too much beta-carotene rich foods like carrots, squash or sweet potato, but was a skin symptom of hypothyroidism.  

Two days ago, I posted the photo below on social media. I now understand the significance of what I wrote;

“Update from A Dietitian’s Journey – Part II: It’s been exactly 2 ½ months since my son’s wedding and 2 months since I began thyroid treatment. I think what is most noticeable is that the yellowish skin colour is gone.”

I now know this was carotinemia which has recently resolved —  between the photo of last week (August 17, 2022) and this week (August 24, 2022).
 

 

How my Clinical Practice is Impacted

Just as my clinical practice changed 5 years ago when I came to understand what hyperinsulinemia was, and how early clinical signs of developing type 2 diabetes are evident as long as 20 years before diagnosis, it is changing again as a result of what I am learning about hypothyroidism.

Understanding the wide range of clinical and subclinical symptoms that people may have leads me to ask additional questions, to look at lab test results differently, and to ask for additional ones if it seems clinical warranted. While it is beyond the scope of practice of a Dietitian to diagnose any disease or to treat hypothyroidism, I am more aware of what to look and this helps me to refer people back to their doctor if I feel there may be a clinical concern.

Final Thoughts…

The list of skin symptoms in hypothyroidism in this article is by no means exhaustive.  There are others discussed in the literature that present, particularly as the disease progresses.  Since the goal of this article was to present symptoms that may present early or with advancing hypothyroidism, additional symptoms are beyond the scope of this article.

If you think that you, or someone you know may have symptoms of hypothyroidism, please consult with a medical doctor. 

More Info

If you would like more information about the services I provide people who are newly diagnosed with hypothyroidism, please send me a note through the Contact Me form, above.

To your good health!

Joy

 

You can follow me on:

Twitter: https://twitter.com/JoyKiddie
Facebook: https://www.facebook.com/BetterByDesignNutrition/

 

References

    1. American Thyroid Association, Prevalence and Impact of Thyroid Disease, https://www.thyroid.org/media-main/press-room/, accessed August 26, 2022
    2. Kasumagic-Halilovic E. Thyroid Disease and the Skin. Annals Thyroid Res. 2014;1(2): 27-31.
    3. Elshimy G, Chippa V, Correa R. Myxedema. [Updated 2022 May 22]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing. https://www.ncbi.nlm.nih.gov/books/NBK545193/#_NBK545193_pubdet_
    4. Medical News Today, What is Myxedema and How is it Treated, April, 22, 2022, https://www.medicalnewstoday.com/articles/321886
    5. Patterson, JW, Weedon’s Skin Pathology, Cutaneous Mucinoses, Elsevier Canada; 5th edition (April 20, 2020)

 

Copyright ©2022 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

Measure of Health With a New Diagnosis – a Dietitian’s Journey

This article is the second entry in A Dietitian’s Journey Part II, which began with my recent diagnosis of hypothyroidism and is about how I now measure health due to my diagnosis.

NOTE: Articles posted under A Dietitian’s  Journey are separate from referenced clinical articles (categorized as Science Made Simple articles) because these are about what happened to me (i.e., anecdotal) and based on my personal observation.

DISCLAIMER: The information in this post should not be taken as a recommendation to self-diagnose, self-interpret diagnostic tests, or self-treat any suspected disorder. It is essential that people who suspect they may have symptoms of any condition consult with their doctor, as only a medical doctor can diagnose and treat.

A Dietitian’s Journey – Part I

“A Dietitian’s Journey” was my personal weight-loss and health-recovery journey that began on March 5, 2017 when I decided to make dietary and lifestyle changes so that I could reclaim my health. At that time, I was obese, had type 2 diabetes for the previous 8 years, and extremely high blood pressure.  I achieved my goal two years later, on March 5, 2019. In retrospect, I realize why it took a year longer than I anticipated.  It is because I had high TSH levels, almost out of range. I had borderline subclinical hypothyroidism.

I believe that you can’t achieve a goal you don’t set“.  In other words, I accomplished my health goals the last time because I set them. As the popular expression goes, “A goal without a plan is a wish.”

I wanted to achieve a normal body weight, be in remission of both type 2 diabetes and hypertension (high blood pressure).

Two years later, on March 5, 2019, I accomplished all but one of my goals, and the last one I achieve 3 months later. I lost:

    • 55 pounds
    • 12- 1/2 inches off my waist
    • 3 -1/2 inches off my chest
    • 6 -1/2 inches off my neck
    • 4 inches off each arm
    • 2- 1/2 inches off each thigh
    • I met the criteria for partial remission of type 2 diabetes 3 months earlier
    • my blood pressure still ranged between normal and pre-hypertension

If you want to get an idea of what I actually looked like at the beginning and at the end, there are two short videos on my Two Year Anniversary post that tell the story well.  The first video was taken when I started my journey, and it is very apparent how obese I was, and how difficult it was for me to walk and talk at the same time. The second clip was taken when I completed my journey and the difference is unmistakable.  

After recovering from Covid, I began hiking, and posted this encouraging “mountain top experience” post as my 5-year update. That was the pinnacle of recovering my heath. 

Except for the ~20 pounds that I gained over the past 2 years (like most others during Covid), my weight has been stable. I continued to remain in partial remission of type 2 diabetes, and my blood pressure was normal until this past December.  In retrospect, that is when my health began to change. 

A Dietitian’s Journey – Part II

As told in last week’s post which was the first entry in Part II of A Dietitian’s Journey), things didn’t go as planned. Here is an excerpt from that post;

“Despite having had both vaccines (April 2021, July 2021), in March of 2022, I came down with what my doctor assumed was Covid again. At first, the symptoms were pretty much the same as in August 2020, muscle aches and joint pain, being exhausted, feeling cold all the time and my lips were frequently blue, but I did not have a headache.  I was loaned an oximeter by a family member who is a nurse and I found it quite strange that my body temperature was always two degrees below normal even though I had fever-like symptoms of being cold and shivering.  The muscle aches were significant, as was the fatigue, but since these are also symptoms of Covid, I didn’t think much of it.  It was only when I began to develop symptoms that were not associated with Covid that I began to become concerned.”

When I saw my doctor last Friday, he thought that it was very likely I had hypothyroidism, but wanted to run some lab tests to rule out any other possibilities.  I went to the lab last Monday morning, and my results came back late in the day. The ones I was waiting for showed exactly what both my doctor and I expected they would based on the supplements I had been taking prior to seeing him.  What I didn’t expect was that my blood sugar would indicate that I was no longer in partial remission of type 2 diabetes.  My blood pressure was higher than it had been in many years in his office, so I began taking it several times a day to see if it was “white coat syndrome” or genuinely high.  Unfortunately, it was the latter.  I knew what I had to do.  I sent him a fax, reported my blood pressure readings, and asked if he thought it was warranted, that he call in a prescription for the same medication I was on 4 years ago.

Last week I did quite a bit of research to better understand how low thyroid hormones could contribute to my high blood sugar and high blood pressure  — despite me continuing to eat a low carb diet. I wrote this referenced article about the metabolic changes that occur due to hypothyroidism that explains how thyroid hormones act on every organ system in the body, and as a result of hypothyroidism, there is a slowing of metabolism which results in weight gain, high cholesterol, high blood sugar and high blood pressure.  Now it was making sense.

I knew one of the symptoms of hypothyroidism was “weight gain,” but I had no idea that it could occur over such a short time frame! Two months ago at my youngest son’s wedding, I looked like I did when I was 55 pounds heavier!

As described in last week’s post, I was very sick but it was devastating to look  like I did! Today my appearance is almost back to normal. 

Sometimes we have to look beyond what something looks like to the timeframe over which it occurred.

Following Up With my Doctor

Today I had my follow-up appointment with my doctor where we reviewed my lab test results from last week, and discussed next steps. My doctor requisitioned a free T4 test to see how my body is responding to the thyroid hormone treatment that he is overseeing.  He also gave me a requisition for a Thyroid Peroxidase antibody (TPO) test to find out if I have Hashimoto’s disease or if my hypothyroidism is due to my past thyroid surgery for a benign tumour. This article from my long-standing dietetic practice explains what these are.

Since Hashimoto’s is an autoimmune disease, how I would choose to approach my diet if the results of that test are positive would be different than if it comes back negative. 

I should have the results back tomorrow or Monday, but in the meantime, I am thinking about what I will do to recover my health once again, and how I will measure my success.

Once again, I am asking myself “what does success look like,” but this time it is in the context of this new diagnosis.

From what I have read, it is possible for my blood sugar and blood pressure to return to normal once the doctor adjusts my thyroid hormone replacement to its optimal dose, however for this goal to be “measurable” I need to have a better idea of how long this could take. 

A Dietitian’s Journey continues…

To your good health,

Joy

You can follow me on:

Twitter: https://twitter.com/lchfRD
Facebook: https://www.facebook.com/BetterByDesignNutrition/

 

Copyright ©2022 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

Symptoms of Hypothyroidism Mistakenly Blamed on Aging

DISCLAIMER (August 14, 2022): The information in this post should in no way be taken as a recommendation to self-diagnose, self-interpret diagnostic tests, or self-treat any suspected disorder. It is essential that people who suspect they may have symptoms of any condition consult with their doctor, as only a medical doctor can diagnose and treat.

NOTE: This article contains aspects of my personal story which are clearly marked. My personal experience is not objective data. The pictures are provided only so that people can better understand what the “weight gain” of hypothyroidism can look like, and how different it is from ordinary weight gain. 


In-person visits to the doctor have been minimal over the past two years, and it has been easy for people to discount symptoms such as body aches,  headaches, fatigue, and ‘brain fog’ to having had Covid, or to having ‘long Covid’ [1]. It was only when I began having symptoms that were not consistent with Covid that I began to think that it might be hypothyroidism. You can read my personal account here.

I am not that old, but at the beginning of June (two months ago), our family was in Tofino (Vancouver Island) for the marriage of my youngest son. The groom’s eldest brother assumed that my inability to walk on the sand, up the path to the hotel, or get up from a chair was a result of me having “aged.”

He had no idea that I was hiking in North Vancouver and Golden Ears Provincial Park for several hours at a time last summer. I knew that it was abnormal for me to feel so exhausted and for my muscles to feel so weak, and one look in the mirror told me something was very wrong.

In a matter of just a few weeks, I went from looking as I have the last two years to looking as I did when I was 55 pounds overweight. For the sake of this special occasion, I said nothing to my family, but was very concerned for my health.  It was also exceedingly hard for me to be in family photographs that I knew would be viewed for years to come.

I planned to contact my doctor when I returned home and have him assess me to determine whether I had what I suspected was hypothyroidism. 

Last Friday, my doctor confirmed that my symptoms were consistent with that diagnosis. I was surprised when he said that it was not unexpected in light of my lab work over the previous nine years, my past thyroid surgery many years ago, and my having experienced periodic hypothyroid symptoms since that time. Unfortunately, it took almost a decade for me to get diagnosed because of the limitations placed on doctors regarding which tests they can requisition under what circumstances. 

Common Hypothyroid Symptoms May Often be Assumed to be Aging

from https://www.thyroid.org/thyroid-disease-older-patient/

People assume that it is normal for ‘older adults’ to have body aches, joint pain, fatigue, feel chilled when others do not, experience constipation, have dry skin or hair loss, be forgetful, or to even experience depression. However, these are NOT typical signs of aging but ARE common symptoms of hypothyroidism. 

The above-mentioned symptoms are so non-specific that many would not give them a second thought. An older person who is already limited to a one-issue-ten-minute remote doctor’s appointment would likely be hesitant to book a phone call to discuss these symptoms with their doctor. After all, they would conclude, these could be the result of so many different things, or “just the normal effect of aging”. 

Consider constipation as an example. Chronic constipation affects 15% of adults and is the sixth most commonly reported GI symptom [3]. Within the context of a lack of mobility that we have all faced due to lockdown restrictions, how many people would give increased constipation a second thought?

Consider mood changes as another example. It is well-documented that the social isolation associated with the pandemic lockdowns has taken a toll on the mental health of people of all ages. It is easy to attribute symptoms of  decreased cognitive function, forgetfulness, or even depression in older adults to increased social isolation rather than considering a diagnosis of hypothyroidism.

Symptoms such as loss of hair on the legs or arms may be attributed to the natural process of aging, and while it is normal to have less hair on the arms or legs as people age, it is not normal to lose all the hair. Although no longer needing to shave or wax one’s legs may be perceived as a benefit of aging (like no longer having a ‘period’ after menopause), a complete loss of hair on the legs or arms is something that is not a normal part of aging. Symptoms like these should be brought to the attention of one’s doctor. Likewise, while it may be nice for someone not to feel as sweaty in the heat of the summer as one did when they were younger, sweating is how humans stay cool, and decreased sweating can be dangerous! 

Symptoms of constipation, hair loss on legs and arms, decreased sweating, forgetfulness, and mood changes such as depression are not part of “aging” but are symptoms that one’s doctor should assess.  

Untreated Hypothyroidism can be Dangerous

Myxedema describes advanced hypothyroidism that occurs when the condition is left untreated or inadequately treated [4]. This term is also applied to hypothyroidism’s effects on the skin, where it looks puffy and swollen and takes on a waxy consistency [4]. 

Below is a photo of what I looked like hiking 3 months before my son’s wedding, what I looked like with myxedema at his wedding, and what I look like today. I don’t share these photos easily because my son’s wedding was a special occasion, and not only did I look and feel terrible, in retrospect, I was very unwell. I am sharing them so that people can understand what the edema / myxedema of hypothyroidism looks like and how quickly it can progress, and how serious hypothyroidism can be if left untreated or undertreated.

Myxedema of hypothyroidism is very different from ordinary weight gain. I hope that by sharing these photos people will be better equipped to recognize this symptom in themselves or in others, and ensure that medical attention is sought. 

Getting Diagnosed

Each province in Canada sets its policy for provincial medical plans covering laboratory tests. In the US, which testing is covered is determined by whether they are performed by in-network or out-of-network labs. 

In British Columbia, thyroid testing covered by the provincial health plan is determined by a 2018 document titled Thyroid Function Testing in the Diagnosis and Monitoring of Thyroid Function Disorder [2]. These guidelines outline testing for thyroid stimulating hormone (TSH), free thyroxine (fT4), free triiodothyronine (fT3), and anti-thyroid peroxidase (TPO).  

Unless someone has specific risk factors for thyroid disease (older age, strong personal or family history of thyroid disease, taking drugs such as lithium (used in bipolar disorder) or amiodarone (used in cardiac dysrhythmia), or grew up in a developing country known to have either iodine excess or deficiency),  individuals are required to exhibit several of the specific symptoms listed below to even qualify for thyroid hormone testing. 

The problem is that typical symptoms such as cold intolerance,  edema,  decreased sweating, and skin changes often don’t appear until much later in the progression of the disease.  Moreover, in some individuals, these symptoms do not appear at all.

Furthermore, as outlined in the previous article, even if a person meets the criteria for a TSH test, the results would need to come back significantly higher than the cutoffs to qualify for a free T4 (fT4) or free T3 (fT3) test. 

People here and in other places with similar policies have no choice but to live with many symptoms documented to be associated with hypothyroidism but outside narrowly defined diagnostic criteria until they become sick enough to warrant testing. 


NOTE: these photos are for illustrative purposes only. 

[LEFT: me hiking March 5, 2022.  MIDDLE: me at my youngest’s son’s wedding on June 3, 2022, only 2 months ago. RIGHT: Me today (August 8, 2022), only two months after my son’s wedding with 75% of the edema resolved.

UPDATE [August 25, 2022] The photo on the left was taken 2 ¾ months ago. The photo on the right was taken today, 2 months after beginning treatment for hypothyroidism. They are provided only as an illustration of what symptoms can look like and how quickly they can resolve with medical treatment. (I deliberately left the lines marking the hairline and chin to make comparison easier.) 

LEFT: before diagnosis and treatment RIGHT: 2 months after diagnosis and starting treatment [for illustrative purposes only]

The photos below are of my left leg without edema and with it. While my legs are still ‘waxy’ looking from the myxedema, the extreme swelling resolved within a few days of beginning thyroid hormone replacement. This photo is for illustrative purposes only and does NOT provide any clinical information.

While each person may exhibit different symptoms, this is fairly typical of the length of time over which the “weight gain” of hypothyroidism can occur, and also the time-frame over which it can resolve with treatment.


It is important to understand that untreated hypothyroidism can progress and the results of a myxedema crisis which can be fatal. The death rate for a myxedema crisis is between 20-60%, even with treatment [5]. 

A myxedema crisis is often incorrectly called a ‘myxedema coma,’ but this term is misleading since the person rarely experiences a coma.

The most noticeable feature of a myxedema crisis is the person’s significant deterioration in mental function [5]. The slowness of thought, decrease in attention, and apathy can easily be confused with symptoms of depression[6], but in severe untreated hypothyroidism, people can exhibit significant agitation and even psychosis and paranoia, referred to as “myxedema madness” [6]. In addition, there have been cases reported in the literature of people hospitalized with suspected affective (mood) disorders such as bipolar disorder– even psychosis that turned out to be a myxedema crisis and that resolved with thyroid hormone treatment [7].

A myxedema crisis may occur because someone had untreated hypothyroidism. It can also happen because someone stopped taking their medication or was taking an incorrect dosage. Therefore, being correctly diagnosed, treated and followed by a physician is essential.

I found the following explanation from a recent article on hypothyroidism [8] very helpful as it explains how different people with the condition may have various symptoms.

“It is important to maintain a high index of suspicion for hypothyroidism since the signs and symptoms can be mild and nonspecific and  different symptoms may be present in different patients. Typical features such as cold intolerance,  puffiness,  decreased sweating and skin changes may not be present always. Inquire about dry skin, voice changes, hair loss, constipation,  fatigue, muscle cramps, cold intolerance, sleep disturbances,  menstrual cycle abnormalities,  weight gain, and galactorrhea  (nipple discharge not associated with lactation / breastfeeding). Also obtain a complete medical, surgical, medication, and family history” [8].

Note (August 15, 2022): Over-treatment with thyroid hormones also poses a risk of thyrotoxicosis, or “thyroid storm,” outlined in this newer article.

Final Thoughts…

By virtual of their age, older adults in British Columbia qualify for thyroid testing. If older people exhibit even a few of the common symptoms of hypothyroidism, such as long standing body aches or joint pain, unexplained fatigue, feeling usually chilled, constipation, dry skin or hair loss, forgetfulness or depression, this should be brought to their doctor’s attention. These are not typical signs of aging but are common symptoms associated with hypothyroidism. 

For younger individuals without preexisting risk factors and that do not have the specific symptoms listed on the diagnostic criteria, the reality is that they do not qualify for testing. Unfortunately, they will need to get quite unwell before they are able to be diagnosed and treated, and their doctor’s hands are tied by a system that will not enable them to test T3 or T4 — even in the presence of high-normal TSH, or symptoms known to be associated with hypothyroidism, but not on the diagnostic criteria list.

Surely, there has to be a way that people can be tested, but that does not put additional financial strain on an already overtaxed public healthcare system?

Currently people have two alternative options;

(1) pay significant out of pocket costs to see a Functional Medicine MD or Integrative Health MD where they can be properly diagnosed and treated.

(2) pay a naturopath added costs for them to requisition thyroid tests, but one concern is since they are not medical doctors, they do not have the training to rule out liver, kidney or heart disease that can mimic many of the same symptoms as hypothyroidism, and that requires medical attention.

In the previous post, I mentioned the option of enabling patients to self-pay at the same cost as the government pays for TSH, T3 and T4 tests. This way if the lab tests results come back abnormal, their doctor can oversee both diagnosis and treatment (or refer them to an endocrinologist).

In British Columbia, someone can pay (at government rates) $9.90 for a TSH test, $12.12 for a free T4 test, or T4 or total thyroxine test, and pay $9.35 for a free T3 test [9]. Under the current system, the government only applies a lab volume discount (based on 2011-2012 volumes) for some fee-for-service (FFS) tests [10] so under this model, only 38% of tests are reimbursed at 100% of the published fee, whereas 62% are only reimbursed at 50% of the published fee [3].  It is not clear from the government publication which rate applies to thyroid testing, but even if people are required to pay 100% of the costs, the total cost of a TSH test, and a free T4 test, and a free T3 test is just over $30.  

I was disheartened to learn recently that even if patients are willing to pay the full cost of thyroid testing, their doctor is under no obligation to write the lab requisition. This is because licensing requirements require doctors who write a lab test requisition to also take responsibility to oversee care based on those results. Unfortunately, not all doctors are willing to treat those with subclinical hypothyroidism. 

How I May be Able to Assist

I currently assist my clients in requesting that their doctor refer them to an allergist if I believe there is clinical reason to suspect IgE mediated food allergies. I also will request that a doctor requisition a fasting insulin (or c-peptide test) along with a fasting glucose if based on assessment I have reason to suspect a person’s pancreas may be working too hard to keep fasting blood glucose and HbA1C in the normal range. In the same way, if I have clinical reason to be concerned about a person’s thyroid function, I will request that a doctor requisition thyroid testing. These requests are by no means a guarantee that a person’s doctor will agree to requisition blood tests, but it has been my experience that when clinical concerns are documented, most doctors are willing investigate further. 

More Info

If you would like more information about the services I provide people who are newly diagnosed with hypothyroidism, please send me a note through the Contact Me form, above.

To your good health!

Joy

 

You can follow me on:

Twitter: https://twitter.com/JoyKiddie
Facebook: https://www.facebook.com/BetterByDesignNutrition/

 

References

    1. John Hopkins Medicine, Long COVID: Long-Term Effects of COVID-19, June 14, 2022, https://www.hopkinsmedicine.org/health/conditions-and-diseases/coronavirus/covid-long-haulers-long-term-effects-of-covid19
    2. BC Guidelines & Protocols Advisory Committee, Thyroid Function Testing in the Diagnosis and Monitoring of Thyroid Function Disorder, October 24, 2018
    3. Bharucha AE, Lacy BE. Mechanisms, Evaluation, and Management of Chronic Constipation. Gastroenterology. 2020;158(5):1232-1249.e3. doi:10.1053/j.gastro.2019.12.034
    4. Medical News Today, What is Myxedema and How is it Treated, April, 22, 2022, https://www.medicalnewstoday.com/articles/321886
    5. Elshimy G, Chippa V, Correa R. Myxedema. [Updated 2022 May 22]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing. https://www.ncbi.nlm.nih.gov/books/NBK545193/#_NBK545193_pubdet_
    6. Samuels MH. Psychiatric and cognitive manifestations of hypothyroidism. Curr Opin Endocrinol Diabetes Obes. 2014;21(5):377-383. doi:10.1097/MED.0000000000000089
    7. Heinrich TW, Grahm G. Hypothyroidism Presenting as Psychosis: Myxedema Madness Revisited. Prim Care Companion J Clin Psychiatry. 2003;5(6):260-266. doi:10.4088/pcc.v05n0603
    8. Patil N, Rehman A, Jialal I. Hypothyroidism. [Updated 2022 Jun 19]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing;
    9. Government of British Columbia, Ministry of Health, Schedule of Fees for Laboratory Services – Outpatient, Payment Schedule, revised April 1, 2022, http://www.bccss.org/bcaplm-site/Documents/Programs/laboratory_services_schedule_of_fees.pdf
    10. BC Agency for Pathology and Laboratory Medicine (BCAPLM), Outpatient Payment Schedule, Laboratory Volume Discounting (LVD), http://www.bccss.org/clinical-services/bcaplm/health-professionals/outpatient-payment-schedule

 

Copyright ©2022 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

 

When a New Diagnosis is a Long Time Coming

Three weeks ago, I wrote an article  about how a diagnosis of hypothyroidism is made and why it takes until someone has been unwell for quite a while before they are finally diagnosed.  In one sense, that article laid the foundation for this one.

DISCLAIMER: This article a personal account posted under A Dietitian’s Journey. The information in this post should in no way be taken as a recommendation to self-diagnose, self-interpret diagnostic tests, or self-treat any suspected disorder. It is essential that people who suspect they may have symptoms of any condition consult with their doctor, as only a medical doctor can diagnose and treat.

Two years ago, in the summer of 2019, I was feeling fantastic and was in remission of type 2 diabetes and hypertension and was celebrating my “little black dress moment.”

In August 2020, I had what my doctor assumed was Covid (back pain, non-stop headache, and couldn’t stop shivering) and since at that point the line up for a nasal swab was 6 hours long due to one of the testing sites closing, my doctor recommended that I simply assume I was positive, and self-isolate for two weeks, which I did.

For many weeks afterwards, I had overall muscle pain and weakness, as well as tingling and numbness in my fingertips, what is referred to as “brain fog”, and unbelievable fatigue. I went from being reasonably active and fit in the spring, to finding it difficult to even walk up or down a flight of stairs by August. Covid was new at that point, so none of us knew what to expect, but it took months until I began to feel reasonably normal. I learned to live with the muscle aches, joint pain, ‘brain fog’, and fatigue. The joint pain persisted for a long time, and was assumed to be post-viral arthritis as I had this once before when I had rubella as an adult.

Despite having had both vaccines (April 2021, July 2021), in March of 2022, I came down with what my doctor assumed was Covid again. At first, the symptoms were pretty much the same as in August 2020, muscle aches and joint pain, being exhausted, feeling cold all the time and my lips were frequently blue, but I did not have a headache.  I was loaned an oximeter by a family member who is a nurse and I found it quite strange that my body temperature was always two degrees below normal even though I had fever-like symptoms of being cold and shivering.  The muscle aches were significant, as was the fatigue, but since these are also symptoms of Covid, I didn’t think much of it.  It was only when I began to develop symptoms that were not associated with Covid that I began to become concerned.  One of those symptoms was non-pitting edema in my lower legs and feet, and I don’t mean just a little bit of swelling. Below is a picture of before, and during;

I ordered compression stockings on-line and wore them daily to help keep the swelling down, but carried on working and writing the book, even though I was very tired all the time. I also began to have a very weird sensation in my mouth – my tongue became enlarged, and the salivary glands under my tongue were swollen. Since both of these affected my sense of taste, I thought this may be related to Covid, but then it progressed to the point where I found it difficult to talk properly because my tongue seemed too big for my mouth. I also began losing hair, but this had occurred several years ago, too.  At the time, my TSH was “in the normal range”, so no further testing was done (see this article to know why TSH alone is not good indicator of hypothyroidism, especially when it is at the high end of the normal range, which mine was).  In retrospect, the subclinical problem with my thyroid has been going on quite a while. Sometimes it would be worse than others, which is not unusual.

Fast forward to two months ago (beginning of June), which was my youngest son’s wedding. I was still experiencing fatigue and muscle aches, chills that would come and go, would get bluish lips, and continued to have significant (non-pitting) edema in my legs and ankles, and was wearing compression stockings all the time — even at the wedding. The skin on my cheeks had become flaky and dry and despite trying multiple types of intense moisturizers, nothing helped. My mouth symptoms had progressed to the point that I found it difficult to say certain words when speaking with my clients because my tongue seemed too large for my mouth, and the salivary glands underneath my tongue were swollen. I continued to have overall muscle aches and weakness, but it had slowly progressed to the point where it was difficult for me to get up from a chair, or to get out of my car without pushing myself up with my hands.  I was wondering if I had some form of “long-Covid,” but what got me starting to think that my symptoms had something to do with my thyroid was the very noticeable swelling in my face. At my son’s wedding I looked like I did when I was 55 pounds heavier, but without significant weight gain.

After doing some reading in the scientific literature, as well as chatting with a couple of functional medicine doctors, I began to think that my symptoms were consistent with hypothyroidism.  In addition, I knew that when I was in my early 20s I had a benign tumour removed from the isthmus of my thyroid and as part of the pre-surgery work up, I had an x-ray that required me to drink radioactive iodine. It wasn’t known at the time but it is known now that both the surgery on the thyroid (even though it remains largely intact), as well as the exposure to high doses of radioactive iodine can initiate a process that can lead to hypothyroidism years later.

It is also apparently possible that having had Covid back in 2020 may have initiated it and/or it may have been initiated as a response to the having the vaccines. I am not blaming either the virus or the vaccines because my thyroid surgery and exposure to high doses of radioactive iodine predated this by decades, but they may have been the precipitating event to symptoms.  It is also possible that symptoms would have started on their own simply as a result of age.

I knew I was unwell and needed to see my doctor in person. After my son’s wedding, I called his office and wanted to go in and have him assess me for hypothyroidism, but he was out of town. Instead of meeting with the locum, I decided to wait until he was back. In the meantime I began using some supplements that are involved in thyroid metabolism, such as kept (for iodine), selenium and some other nutrients and while they helped a little bit, it was not significant. After doing a great deal of reading in the literature and listening to several medical presentations by a well-known endocrinologist and professor of medicine from the US, I decided while waiting to see my doctor that I would try using a very small amounts of another type of supplement to see if it made any difference in my symptoms. I introduced it at half the rate and half of the dose usually used because (1) I had not yet seen my doctor (was not under medical supervision yet) and (2) I was aware that use of this supplement was not something to be taken lightly as it can cause problems for older individuals, or those with heart disease (which I don’t have). 

This morning I saw my doctor for the first time since Covid began. I had sent him a fax last week outlining the ways I had improved because I knew it was too much information for a 10 minute visit. I explained that I was feeling significantly better. My face swelling had gone down a great deal, the edema in my legs had almost disappeared – to the point that I could walk around bare-legged in the excessive heat we had last week with NO swelling what-so-ever. The skin on my legs is still very tight and shiny, but no edema. I lost 5-6 pounds of water-weight (face, legs and abdomen) and most noticeable, the muscle weakness is gone!  I could walk up and downstairs, carry heavy parcels, and can get up from a chair or out of my car with ease.  I also explained in the fax that I rarely feel cold, but still have occasional blue lips and chills late in the afternoon, but that from what I’ve read in the literature, many people do better on the same amount split over 3 doses, rather than two. 

When my doctor entered the examining room, he said he had just re-read the fax and based on what I wrote, he thinks it is very likely that I have hypothyroidism, but he wants to rule out other things that could look like it and aren’t, or that mimic it. He wasn’t in a rush, like he usually is. He looked at the pictures I had on my phone —ones I had taken of my legs, my tongue, my face. When he saw the picture of me two months ago at my youngest son’s wedding, he simply said “oh my.” He then gave me a very thorough examination.  He palpitated my thyroid and listened for a long time to my heart and lungs.  After examining me, he pointed out several other physical symptoms that I have that are quite consistent with hypothyroidism, and said “Joy, I think your conclusion is right on.” I was somewhere between shocked and elated.

My doctor then brought up my past lab work on his screen and remarked that my TSH has been “high normal” since 2013 (see below), and that I often had low ferritin with no explanation, as well as past “unexplained” issues with hair loss.  I had nine years with subclinical symptoms but no testing could be done because as indicated on the lab test results below “The free T4 was cancelled. The protocol recommends no further testing.

TSH – 2013 – “in normal range”
TSG – 2015 – “in normal range”

I mentioned to him that I wondered what the results would have shown if my T3 or T4 were tested in 2013, or 2015, when my TSH was high-normal. He replied “unfortunately, unless someone has clear symptoms that are consistent with hypothyroidism there is nothing we can do, but your symptoms are very consistent now, but I think this diagnosis was a long time coming.” Surprisingly, we saw eye to eye.

I think my doctor realized that the guidelines being as they are means that people like me have to get quite unwell before they are finally diagnosed and treated.  I realized that his hands were effectively tied by a system that will not enable him to test T3 or T4 even with high-normal TSH, without overt symptoms. He could do nothing until I got much sicker. 

I was delighted by his response. He has been my doctor for 20 years and was not receptive to my use of a low carb and then a ketogenic diet to put my type 2 diabetes into remission, and previously refused twice to test my fasting insulin, along with my fasting blood glucose.  Today he was very different.

When I asked if he was going to refer me back to the endocrinologist I used to see when I was diabetic and have her manage my thyroid replacement medication and he said “No. I don’t believe in changing something that is clearly working. I want you to keep taking what you’re taking in the same amount you are now, and I am going to run some lab work to see if you have gotten the amount right. We may need to increase it a little or change the timing to address the late afternoon chills, but no, I’m not going to “fix” something that is no longer broken.” He even agreed to add a fasting insulin test, without any protest!

I don’t know what happened to make my doctor change his mind and how he approaches these types of matters, but today I said to him that it has been a long time since I was this delighted with his approach, and that I am very thankful that he is my doctor because he practices good medicine. I offered him my hand and he shook it warmly and thanked me.

I guess if I can change how I practice dietetics based on new evidence, so can my doctor — or your doctor.  Don’t give up, or be hesitant to have those difficult conversations with your primary care physician. We need them to oversee our care, and maybe just maybe in the process of interacting with some patients, they learn something they didn’t before, or change because of things they see in their practice. The bottom line was that I needed my doctor to know what I was doing and to examine me and make sure I was not doing something that could cause me harm.  He not only rose to the occasion with grace, but responded in a manner I could have only dreamt of before.

I do not believe that self-treating is ever advisable, and certainly if it were not for Covid and my doctor not having in-person office hours unless it was an emergency, I  would have gone to see him months ago. I am glad I saw him today and am very thankful that he is being so supportive.

I know once we get the levels of thyroid hormones right, that losing the 20 pounds I gained over the pandemic will be possible, but in the meantime, it is no small matter that I got my life back!!

A Dietitian’s Journey continues…

To your good health,

Joy

I don’t post the comparison picture below easily. It is very hard for me to see how bad I looked, but it is important to see just like the leg pictures, above. The photo on the right was taken at my youngest son’s wedding, June 3, 2022 (exactly 2 months ago) at the height of my hypothyroid symptoms.  The photo on the left is a selfie I took today, August 5, 2022, almost exactly two months later. There is still swelling in my face and legs to come down, but any adjustment in thyroid meds only be done after the upcoming lab work.

NOTE (August 15, 2022): It is important to keep in mind that too little, or too much thyroid hormone can have serious consequences.

Untreated or under-treated hypothyroidism can be serious and is when the body gets too little thyroid hormone. This can lead to a myxedema crisis (covered in this article).

Thyrotoxicosis can also be serious and is when the body gets too much thyroid hormone. This can occur in untreated hyperthyroidism, or by self-treating hypothyroidism (covered in this article).

If you suspect you may have hypothyroidism (or any other clinical condition), consult with your doctor, and “don’t try this at home.”
 

You can follow me on:

Twitter: https://twitter.com/lchfRD
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Copyright ©2022 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

Too Much and Too Little is Killing Us – reducing comorbidities

I just got “the call” that my mother who lives on the other side of the country, has tested positive for Covid-19. She has all of the major comorbidities, so prognosis is not good. Following the news, most of us know that age, obesity, hypertension and diabetes are known to significantly increase the risk of requiring hospitalization and death from Covid-19 (you can read more about that here, here and here), yet all but one of these comorbidities can be put into remission.  We can’t change our age, but we CAN reduce our weight, lower our blood pressure and normalize our blood sugar. To improve our quality of life outside of the pandemic, and to have the best chance of fighting it off if (or more likely when) we get it, we need to put our energy into achieving a normal body weight (and waist circumference), blood pressure and blood sugar now.

I am from a family of people that always ‘battled with their weight’.

My dad was tall and very fit when he was younger, a boxer and a ski-jumper but as he aged he accumulated excess weight around his middle. He then developed type 2 diabetes, high blood pressure and heart issues and eventually was diagnosed with Alzheimer’s disease, which is sometimes referred to as ‘type 3 diabetes’ by some clinicians. I wrote this article about that when he was first diagnosed.

My dad died just a few weeks shy of his 91st birthday, which is a ‘ripe old age’ of course, but for the last 40 years of his life, he was not in good health. He took several medications due to multiple metabolic conditions related to his diet and lifestyle. Except for his age, many of these conditions could have been put into remission — or at very least, been much better controlled with a change in diet and lifestyle.

My mom will be turning 85 this fall, and has been overweight since I was little. She too has type 2 diabetes and takes multiple medications for various conditions, many related to diet and lifestyle; conditions which could have been greatly improved, if not put into remission with a change in diet and lifestyle. It wasn’t for lack of trying “diets”. When I was young, she weighed her food, counted points and went to “groups” and at times she lost weight, only to put it all back, and then some. Eventually, she stopped trying. I can’t say that I blame her, given what I now know about the drivers to hunger.

Shared comorbidities

When I became overweight and then obese, and developed type 2 diabetes and high blood pressure, I justified that I was at high risk since both of my parents had the same.  I realize now that the “high risk” was our shared diet and lifestyle, more than genetics. Our shared comorbidities were adopted.

I grew up loving to eat good food and unfortunately considered food as both a reward, and a comfort. When someone was happy, we celebrated with good food. When someone was sad, we consoled with “comfort food”.  Food was “medicine”, but not in a good way. It didn’t heal, but contributed to the underlying hyperinsulinemia that drove the disease process. (I’ve written several articles about this topic, but if you only want to read one, I’d recommend this one.)

Those who have followed me for some time know that 3 years ago I began what I call my “journey”.  It took almost two years to do, but I lost ~60 pounds and a foot off my waist and I put my dangerously high blood pressure and uncontrolled type 2 diabetes into remission, as a result.  I recently posted a summary here, to mark my three year health recovery anniversary; two years of active weight loss and a year of maintenance.

Left: April 2017, Middle: April 2019, Right: April 2020

So why am I writing this post?

Like many people, I am upset by this whole “Covid thing” — not just because of my mom being diagnosed. I’ve been following the news, and realize that the hope for a vaccine any time soon is dim, and effective treatments are still lacking. An article published in the journal The Lancet the other day reported that while ~90% of those who have been hospitalized with severe Covid-19 develop IgG antibodies in the first 2 weeks, in non-hospitalized individuals with milder disease or with no symptoms, under 10% develop specific IgG antibodies to the disease.  That means that except for the sickest people who actually survive being hospitalized for several weeks with Covid-19, more than 90% of people who get Covid-19 and recover outside of hospital don’t develop antibodies to this virus [1]. Since the whole purpose to develop a vaccine is to challenge the body to develop antibodies to the virus — the very fact that most of the time people who don’t get that sick don’t develop antibodies, means that the likelihood of most people developing antibodies to a vaccine may be minimal.  As well, in light of this data herd immunity is also a dim prospect because most people that get this disease don’t produce antibodies, which means they aren’t immune and can probably get this virus again.

With little immediate hope of an effective vaccine or of herd immunity, what CAN we do to lower our risk?

I think that we first need to realize that many experts believe it is simply a matter of time until we are all exposed to the SARS-CoV2 virus and develop Covid-19, so we must look at lowering our risk of having a poor outcome. We can’t change our age, which is the biggest risk factor but we CAN do something to change the high risk comorbidities such as obesity, high blood pressure and diabetes.

Too Much and Too Little is Killing Us

For many of us, having both too much and too little is killing us.

We have diets with way too much refined carbohydrate, usually combined with large amounts of refined fat, and our usual diet is full of these in the form of pizza, pastries, take out foods and snack foods.  We now know from a study that was published almost 2 years ago[2] that this combination of both refined carbs and fat results in huge amounts of the neurotransmitter dopamine being released from the reward centre of our brain — way more than when we eat foods with only carbohydrate, or only fat separately. This huge amount of released dopamine results in us wanting to eat these foods, and craving these foods, and being willing to pay more for these foods than foods with only carbs or only fat [2]. Dopamine makes us feel good, and is the same neurotransmitter that is released during sex and that is involved in the addictive ”runner’s high” familiar to athletes. This is one powerful neurotransmitter! It is this dopamine that is driving why so many people on “lock-down” have turned to baking bread and pastries for comfort, and buying snack foods with this same combination once they finally get through a long line to get into a grocery store! 

Too much of these refined “foods” is contributing to 1/3 of Americans and 1/4 of Canadians being obese, and another 1/3 in both countries being overweight. These foods are driving the hyperinsulinemia that underlies many metabolic conditions, including type 2 diabetes and hypertension.  These diseases were killing a great many of us before Covid-19, but knowing that these comorbidities increase our risk of hospitalization and death when we get the virus, why don’t we consider limiting these? I think it is because eating these foods make us feel good and so we self-medicate our stressful lives with something that is socially acceptable. High carbohydrate and fat foods are much more socially acceptable but what will it take for us to see that this for what it is and to consider that we need to change how we eat. If we aren’t willing to admit that our obesity, high blood sugar and high blood pressure are a problem, then maybe we are simply in denial. I certainly was, and wrote about this in “A Dietitian’s Journey”, the account of my own health recovery.

No, I am NOT saying that “all carbs are evil” and I’ve written about this previously, but we need to differentiate between what most of us eat as “carbs” and whole, real food that have carbs in them. I believe we need to eat significantly less of the refined foods that are contributing to our collective weight problem and metabolic health issues, and eat more of the real, whole foods that have gone by the wayside in our diet. I’ve written about the science behind this type of dietary change in many previous articles, and that eating this way is both safe and effective. What I can’t do is motivate people to want to change.

It is my hope that by presenting the evidence, as I have in several recent articles posted on this website that comorbidities such as obesity, hypertension and diabetes are significant risk factors to requiring hospitalization or of dying of Covid-19, that it may motivate some people to consider making some changes. If not now, when — especially given that the hope of a vaccine and/or herd immunity is likely a long way off.

I wish each of you good health and a long life.

If I can help, please let me know.

Joy

You can follow me on:

Twitter: https://twitter.com/JoyKiddie
Facebook: https://www.facebook.com/BetterByDesignNutrition/

Copyright ©2020 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only. The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything you have read or heard in our content.

Reference

  1. Altmann DM, Douek D, Boyton RJ, What policy makers need to know about COVID-19 protective immunity, The Lancet, April 27, 2020, DOI:https://doi.org/10.1016/S0140-6736(20)30985-5
  2. Di Feliceantonio et al., 2018, Supra-Additive Effects of Combining
    Fat and Carbohydrate on Food Reward, Cell Metabolism 28, 1—12

 

 

My Three Year Health Recovery Anniversary — a Dietitian’s Journey

I delayed posting this update to due to the current Covid 19 pandemic, but thought by now we could all use with a little distraction. I hope that this post about my health and weight recovery serves as encouragement as to what is possible simply by eating real, whole food, and sticking with it.

Me – April 2017, 2019 and 2020

Three years ago, on March 5th, 2017 I was sitting at my desk in my office and I didn’t feel well. I didn’t even know what kind of ”unwell” I felt.  I decided to take my blood pressure to see if that would give me a clue.  I was alarmed with the results and decided to lie down and take it again. That didn’t help. Not only was my blood pressure high, it dangerously high.  I was having what is known as a “hypertensive emergency”.  While I hadn’t done so in way too long, I also decided to take my blood sugar. The result was 13.2 mmol/L (238 mg/dl) only a half an hour after I ate, which was way too high — even for someone who had been diagnosed with type 2 diabetes five years earlier.  Here I was, an obese Dietitian with a body mass index (BMI) well over 30, dangerously high blood pressure and blood sugar that clearly showed my type 2 diabetes was not well controlled and I knew that all of these factors put me at significant risk of having a stroke or heart attack. I was scared. Actually I was terrified.

As I’ve said on every podcast I’ve been a guest on, and have written about many times, what I should have done at that point was to have gone straight to my doctor’s office;  even knowing that he would have sent me directly to the hospital by ambulance or taxi due to my dangerously high blood pressure.  I should have gone, let them treat me to get my blood pressure down, including taking the medications they prescribed. Then, with my doctor’s oversight I could have begun a well-designed therapeutic diet to lower all of these significant metabolic markers and in time had my doctor gradually de-prescribed the various medications I would have been given, as my weight, blood pressure and blood sugars normalized.

I didn’t. It was foolish. What I did instead was to immediately change my diet and lifestyle and while I fully acknowledge that this was not a wise choice, that’s what I did.

I was so scared.

In the preceding 6 months, I had two girlfriends die within 3 months of each other; one of a massive heart attack, and the other of a stroke. Both worked in healthcare their entire lives and both had become overweight and had developed some of the same metabolic issues I had. I was terrified because I realized that if I didn’t change, I could be next.

April 2017

That day, I printed off my last set of blood test results, and took all my body measurements as if I were a client. I then designed a Meal Plan for myself as I do for others and from that day on, implemented it ”as if my life depended on it”, because quite literally, it did.

There’s been no looking back! March 5, 2017 was the beginning of my health and weight recovery journey; A Dietitian’s Journey.

April 2018

In the first year, I lost 32 pounds and 8 inches off my waist, and my glycated hemoglobin (HbA1C) no longer met the criteria for Type 2 Diabetes (i.e. was ≤ 6.0 %), and my blood pressure ranged between normal and pre-hypertension. Updated lab work indicated that my triglycerides and cholesterol levels were optimal, however my updated measurements showed that my waist circumference was still not half my height, which is what it needed to be (you can read more about the reason for that here). In addition, my fasting blood sugar remained higher than it should be. I still had work to do. I was in recovery, but not recovered yet.

After consulting with two physician colleagues, I made the decision to lower my carbohydrate intake, and continued to monitor my blood pressure daily and blood sugar several times per day.  I also began doing some resistance training exercises with equipment I had on hand (and that had been collecting dust for years).

April 2017 & April 2019 (same outfit)

After 2 years on my recovery journey, I had lost a total of 55 pounds and 12 inches off my waist but since my blood pressure remained between the pre-hypertensive and hypertensive range, and in discussion with my doctor’s colleague, I decided to go on a ”baby dose” of Ramipril to protect my kidney function. Even though my blood sugar was good and my HbA1C was below the cut-off for type 2 diabetes, my endocrinologist started me on Metformin as a result of my father’s recent diagnosis of Alzheimer’s disease.

I didn’t look at starting on either of those medications as “failure”, as I probably would have been prescribed those at much higher doses from the beginning had I gone to see my doctor March 5, 2017. It was part of my recovery process. My goal however was to make changes so that blood pressure medication would no longer be necessary, but I didn’t know what other changes I could make to have it to come down to a normal level, and for my fasting blood glucose to continue improve as well. After much reading in the scientific literature about circadian rhythms , I realized that to be successful I needed to change when I ate (and didn’t eat) as well as when I was exposed to bright light in order to get my body working according to its natural circadian (24-hour) cycles. I made the changes documented in the literature and began to sleep much better (falling asleep and staying asleep, when I had previously had poor sleep for years). A few months of home monitoring indicated my blood pressure was normal or slightly below and I was getting fasting blood glucose numbers I hadn’t seen before (4.7mmol/L – 5-2 mmol/L). I hadn’t “arrived” but my recovery phase was definitely approaching the end.

A visit to my doctor’s office just before Covid 19 began indicated I had blood pressure that was just below the normal cutoff of 120/70 for someone who is not diabetic, so my doctor de-prescribed the blood pressure medication and recent lab test results indicated that I have completely normal fasting blood sugar [5.2 mmol/L (94 mg/dl)]. Over the past year without trying, I lost another 5 pounds and a little less than an inch off my waist and I am guessing this was probably the result of continued loss of fat balanced by increased weight from added muscle I gained as a result of the intermittent resistance training I was doing.

April 2020

I am now a normal body weight. I have an optimal waist circumference (slightly less than half my height). I am in remission of type two diabetes; both as assessed by fasting blood glucose and HbA1C, and my high blood pressure is in remission. I went from taking 12 different medications three years ago, to leaving my doctor’s office a few weeks ago with one prescription for something non-metabolically related, and a prescription for glucose test strips.

I feel good about myself, about my health and how I look — so much so that in September of this past year I decided to stop straightening my hair and now wear it the way it grows out of my head.  I am “comfortable in my own skin” (and hair) for the first time in almost 3 decades. I didn’t lose weight quickly but it took me many years to become THAT metabolically unhealthy that I gave myself the time I needed to get well and am staying well, without any added effort. The process wasn’t at all difficult to accomplish, or difficult to maintain. All it took was eating real, whole food and reducing the amount of carbohydrate-based foods I ate.  What is nice is that after 3 years on a therapeutic diet, I am now able to add in small amounts of higher carbohydrate-based whole foods into my diet, and tolerate them very well.

While there are many studies showing many others have accomplished similar clinical results as I have eating the same way, doing it myself enables me to encourage my clients because I have “been” there, and I came back!

More Info?

If you would like more information about how I can help you lose weight and keep it off or improve blood pressure, blood sugar or cholesterol please reach out to me. All my services are now provided via Distance Consultation but I already have more than a decade of experience providing virtual nutrition support, so this is nothing new for me. I am licensed as a Dietitian in every province in Canada except PEI and can also provide nutrition education services to those in the US and elsewhere.

You can find more about the details of the different packages I offer by looking under the Services tab or in the Shop. If you have any service-related questions please feel free to send me a note using the Contact Me form above, and I will reply as soon as I can.

To your good health!

Joy

You can follow me on:

Twitter: https://twitter.com/JoyKiddie
Facebook: https://www.facebook.com/BetterByDesignNutrition/

Copyright ©2020 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only. The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything you have read or heard in our content.

 

Five Pounds or Fifty Pounds of Fat – in very real terms

Whether one loses 5 pounds of fat or 50 pounds of fat, I think it is very helpful to visualize just how much that is. Yes, five pounds of fat is much larger than most people realize!

This past week, I purchased a life-sized model of 5 pounds of fat from a well-known nutrition supplier; the same supplier I have purchased life-sized food models from, which I used to use a lot in my practice.  When I received it, I was quite surprised how much room it took up and just how heavy it was.

Here is a photo of the life-sized model 5 pounds of fat on a scale, with my left hand for a size reference:

5 pounds of fat on a scale, with adult hand as reference – © BBDNutrition

Here is a photo of it on an ordinary steno chair:

5 pounds of fat on a steno chair – © BBDNutrition

…and here is 5 pounds of fat being held in my hand:

5 pounds of fat in adult hand – © BBDNutrition

Finally, here is 5 pounds of fat being carried as one would carry an infant:

holding 5 pound of fat – © BBDNutrition

Five pounds of fat is a lot! Sure there is the initial water-loss at the beginning of weight loss, but here I’m talking about fat.

Fat takes up a fair amount of room around one’s waist, or worse inside one’s abdomen or organs. If someone has 20 pounds of fat to lose, that is four of those fat models distributed over their body; legs, belly, arms, neck, back and face and perhaps some in their liver.

I had 55 pounds of excess fat before beginning my journey.

Comparing these two full length photos, it is easy to see how I had the equivalent of one of those fat models over the length of each leg, one distributed between each arm, one distributed over my neck and face and 2 spread out around my waist and hips and some no doubt, in my liver and pancreas. But still, I can’t actually imagine where I was carrying 11 of those, all told!

The fat in my abdomen must have been more than I imagined, as it was wreaking metabolic havoc on my body.  I had very high blood pressure and had type 2 diabetes for 8 years.  You can read the entire story (including lab test results) under “A Dietitian’s Journey” on my affiliate low carb web site by clicking here. Keep in mind that I chose to follow a therapeutic low carbohydrate diet, but there is no one-sized-fits-all diet that is right for everyone.  

Whether you have 5 or 10 pounds of fat to lose, or like me ⁠— a whole lot more, it is really only done a pound or so at a time.  If you have significant amount of weight to lose,  I can not only help you do that, but since I’ve been through it myself, I can encourage you and coach you through it. I provide services across Canada (except PEI) via HIPAA-compliant video conferencing, and most extended benefits providers reimburse for licensed Dietitian services.

More Info?

If you would like more information about the services I offer, please have a look under the Services tab or in the Shop for more information. If you have service-related questions, please feel free to send me a note using the Contact Me form above, and I will reply as soon as I can.

To your good health!

Joy

You can follow me on:

Twitter: https://twitter.com/JoyKiddie
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Copyright ©2020  BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

A Little Black Dress – a personal health and weight loss update

Yesterday I had an occasion to wear a new little black dress that I had bought, and remembered the last time I wore one. Ironically, it was for my Master’s convocation just over 11 years ago, and the dress was a size 16. My degree was in Human Nutrition, yet I was very overweight and had pre-diabetes.

May 25, 2008

The degrees on the wall did not help me understand why ⁠— despite my best efforts to “exercise more and eat less”, I was still overweight.  Despite my research related to the neurotransmitter dopamine, it was not known at the time how dopamine is involved in the potent joint reward system of eating foods that are a combination of both carbohydrate and fat (you can read more about that here). 

I did not understand why following the advice of my physician didn’t help.  I ate according to the (then) Canadian Diabetes Association (now called Diabetes Canada)’s recommendation to eat 65 g of carbohydrate at each meal and 25-45 g of carbs at each snack ⁠— along with lean protein and monounsaturated and polyunsaturated fat and participated in exercise several days each week. I ate “plenty of healthy whole grains” and “lots of fruit and vegetables“, along with low fat dairy,  yet a year later progressed to Type 2 Diabetes; what I was told was a “progressive, chronic disease”.

My studies didn’t help me understand the impact of high levels of circulating insulin on obesity and the effect of the after-meal and after-snack rise in insulin and then it’s drop shortly later on hunger. The reality was, the advice we were taught to “eat less and move more” did nothing to address the underlying issue of being hungry every few hours.  In fact, the detrimental effects of high circulating levels of insulin weren’t taught; only the effects of high blood sugar.

My studies didn’t help me understand that “plenty of healthy whole grains” for someone who is already insulin resistant, with high levels of circulating insulin isn’t helpful.  I didn’t understand how eating plenty of fruit was further contributing to my problems;  both because of it’s high carbohydrate load, as well as it being a high source of fructose. I drank 3 glasses of low-fat milk daily, but didn’t understand the effect of all of those extra carbohydrates on my blood sugar, as well as underlying insulin response.  It was not part of what I studied ⁠— either in my undergraduate degree or Master’s studies, because it simply was not well known.

It is only recently (April 18, 2019) that the American Diabetes Association (ADA) issued their Consensus Report which indicated that “reducing carbohydrate intake has the most evidence for improving blood sugar” (you can read more about that here). In fact, the ADA now includes both a low carbohydrate eating pattern and a very low carbohydrate (keto) eating pattern as Medical Nutrition Therapy for the treatment of those with pre-diabetes, as well as adults with Type 1 or Type 2 Diabetes.

While these are not currently part of Diabetes Canada‘s options, they are recommendations available to those in the United States. In fact, the European Association for the Study of Diabetes (EASD) also classifies low carb diets as Medical Nutrition Therapy and Diabetes Australia released their own updated position paper for people diagnosed with Diabetes who want to adopt a low carbohydrate eating plan. 

Many studies already demonstrate that a well-designed low carbohydrate diet is both safe and effective for the treatment of obesity and Diabetes (see the Physician and Allied Health Provider tab on my affiliate low carbohydrate web site for more information) but much of this has only come to light in the years since I graduated with my Master’s degree.

In the last 4+ years since I first learned about the therapeutic use of a low carbohydrate diet, I have read scores of studies in an effort to become well-informed and continue to do so in order to stay current with the emerging evidence.

April 2017 – April 2019

On March 5, 2017 I began what I have called “A Dietitian’s Journey”. Over the subsequent two years, I put my Type 2 Diabetes into remission, lowered my dangerously high blood pressure and achieved a normal body weight and optimal waist circumference.

You can read my story under A Dietitian’s Journey on my affiliate site.

June 15 2019

I have been in maintenance mode for more than three months and have been able to maintain my weight loss and health gains with little effort.

This photo was taken of me yesterday in my new “little black dress”.

 

The bulk of my Dietetic practice in the past focused on food allergy and food sensitivity (including Celiac disease, Irritable Bowel Syndrome, Inflammatory Bowel Disease), but I am now able to provide a range of options for weight loss and improvement in many metabolic conditions, including Type 2 Diabetes, hypertension and abnormal cholesterol that I was unable to offer a few years ago. I offer variety of evidence-based approaches, including a Mediterranean Diet, a plant-based whole foods approach (vegetarian or including meat, fish and poultry), as well as a low carbohydrate approach (which is what I follow).

If you would like to learn how I can help you, you can learn more about my services under the Services tab or in the Shop.

If you have questions, please feel free to send me a note using the Contact Me form above and I will reply as soon as I can.

To your good health!

Joy

You can follow me on:

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Copyright ©2019 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

 

Green Tea Should not be like Buckley’s®

Recently, I came across a social media post about someone that wanted to drink green tea for it’s health benefits, but just couldn’t get over it’s “bad taste”.  I followed the origin of the thread to Reddit, where people guessed whether green tea’s “off taste” for that person may be genetic, like the taste of cilantro. While that can be the case (i.e. genetic sensitivity to a compound called 6-n-propylthiouracil which is found in some flavonoids), others touched on whether it was because the person was making tea using supermarket green tea bags rather than loose tea, whereas a few people hit on the complexity of the issue.  In this post I will discuss some of the factors that affects whether your green tea has a pleasant or “off taste”,  because after all green tea should be something you actually enjoy and not only drink for it’s health benefits.

NOTE: The first part of this article are some personal details of my experience learning to prepare multi-ethnic food and beverages and the second part of the article is specifically about the preparation of green tea and its health benefits.


Once a Foodie, Always a Foodie

I have been adventurous in trying different kinds of food and beverages since I’m little and I remember my parents taking me to an authentic Japanese restaurant even as a kid.  As a teen, I enjoyed cooking multi-ethnic food and learned authentic Cantonese cooking in the 1970s when my mom took a course in Chinatown. In the 1980’s, I learned authentic Thai cooking from the friend of a family business associate who was from Thailand and in those days one couldn’t buy pre-made Thai curry pastes that are available everywhere now, so I sourced the raw ingredients in Lao-Thai groceries and hand-pounded them myself in a mortar and pestle (that I still own and use!). I still have the recipe books sent to me from Thailand.

It didn’t matter whether it was Asian, Middle Eastern or Jamaican, I was a bit of a purist; wanting the ingredients and cooking method to be as authentic as possible. For me, the best way to find out how to make something was to ask someone from that culture that loved to cook.

What was true about food was also true for beverages.

I couldn’t just enjoy a cup of coffee or glass of wine without knowing more. Whether it was the origin of the coffee beans, the length of time the beans were roasted, or how long the water is in contact with the beans — I needed to know, and I was interested in such things when it was not popular either.

Before “West Coast coffee” was a thing and before there ever was Starbucks® or Peet’s, there was a place called La Vieille Europe on St. Laurent Blvd in Montreal which was where I got my single origin, whole bean coffee. As I found out years later, the son of the roaster that owned that store taught the original roaster from Peet’s in the US how to roast beans. Small world.

When I lived in wine country (Sonoma county) of California for a few years in the early 2000s, I was determined to educate my palate to distinguish between different types of wine, which I did. I knew what I liked — which turned out to be an expensive habit when I returned to Canada after 9/11.  At the beginning I explored the wines of Australia and found some I really liked, but missed the delicious and inexpensive  wines of Sonoma and Napa.

Once again, my palate returned to coffee, but finding a decently roasted coffee in Vancouver BC was harder than I thought. Given that this was the “West Coast”, I was discouraged how difficult it was to find good quality Arabica beans that weren’t over roasted. I stumbled across a few small roasters that did an excellent job, but in time they modified their roasts for “local tastes”, so once again, I was back looking for a new roaster. On a few occasions, I ordered from La Vieille Europe in Montreal because in the 40 or 50 years they have been in business, they never lost their passion for properly roasted, single origin coffee.

Over the 20 years I have lived in Vancouver, I discovered the world of quality tea that is largely unknown to most non-Asian born Chinese. There was one excellent tea importer in the Chinatown that I knew of and one that is still in the Richmond Public market that have single origin estate teas that rival the diversity of the best coffee roaster. Over the past 20 years, I’ve explored different types of tea from China and  have come to like a few; my favourite of which is a fermented tea known as Pu-ehr.

A number of years ago, I stumbled across matcha tea in a specialty Japanese store before it was a “thing”.  Knowing nothing about it, I have since found out that I had been using ‘culinary matcha‘ (designed for making Japanese sweets) for drinking.  No wonder it tasted bitter and I needed to blend it with other ingredients to make it palatable. Thankfully, when fresh it had the same health benefits, which I wrote about in 2013 in this article about the Role of Green Tea Powder (Matcha) in Weight and Abdominal Fat Loss. As you’ll read below, I have since learned about making and enjoying real ceremonial-grade matcha, which is intended for drinking from large matcha bowls.

Learning about Japanese Green Tea

At the beginning of this year, I began to explore green teas from Japan when I discovered Hibiki-An, an online tea importer from Uji region of Kyoto. My culinary world expanded once again.

Unable to decide between the many different types and grades of tea that they carry, I order a sampler of 3 types of green teas (Sencha, Gyokuro Superior and Sencha Fukamushi).  They came in 4 oz individual bags — the quantity that can be reasonably be used up within 3 months, when it is fresh.  All 3 teas were all of “superior” grade, which is not the best quality (as my palate is not developed yet) but is a high grade tea.

When the tea arrived, it came with very specific brewing instructions (a summary of the much more detailed instructions on their web page). I’ve since learned that different types of green tea require different water temperatures and different lengths of brewing time.

Wow, who knew?

For the purpose of “cooling” the water to just the right temperature, there is a yuzamashi — which is a small ceramic cup with a spout that the boiled water gets poured into to cool momentarily before being poured into the kyuzu; a special tea pot with a single handle, built in mesh filter and large opening for the water (see photo, above).

You don’t need the get fancy, though.  I had these things for years from my days exploring different regional teas, but one can use an ordinary bowl to cool the water and any plain ceramic tea pot to brew the tea in!

Tea to Water Ratio, Water Temperature and Steeping Time

Each type of green tea has a very specific ratio of green tea leaves to water, and very specific water temperatures and steeping time.

For example, of the three teas in my sample set, Sencha is brewed at 80° Celsius (176 ° Fahrenheit) for one minute, Gyokuro is brewed at 60-70 ° Celicus (140-158° Fahrenheit) for 1 -1/2 to 2 minutes and Sencha Fukamushi is brewed at the same temperature as regular Sencha, but for only 40-45 seconds.

I’ve discovered that following these guidelines using good quality, fresh tea leaves makes a cup of tea that is like nothing I’ve tasted anywhere before. It is not simply snobbery, but the science of what makes for a good cup of tea.

Note: I downloaded several studies that have researched the difference in brewing time, water to tea leaf ratio and water temperature but have decided against boring anyone with the details.

Recently, I became ready to move onto “realmatcha tea and ordered some from the same supplier in Japan.

It came in tiny cans (quantities that should be used up in a 3 week period).

The colour was a bright jade green and the taste had no hint of bitterness whatsoever!

It tastes amazing!

My teas ordered from Japan are my “weekend teas” and during the week I used run-of-the-mill Sencha purchased locally at a Japanese store.

I drink them because I like them and for the health benefits.

Health Benefits of Green Tea

The health benefits of green tea are many. Several large-scale population studies have linked increased green tea consumption with significant reductions in the symptoms of metabolic syndrome; a cluster of clinical symptoms which include insulin resistance and hyperinsulinemia (high levels of circulating insulin), Type 2 Diabetes, high blood pressure, and cardiovascular disease including coronary heart disease and atherosclerosis.

Catechins make up ~ 30% of green tea’s dry weight, of which 60—80% are catechins. Oolong and black tea which are produced from partially fermented or completely fermented tea leaves contains approximately half the catechin content of green tea

It is believed that epigallocatechin gallate (EGCG) which is the most abundant catechin in green tea actually mimics the action of insulin, which has positive health effects for people with insulin resistance or Type 2 Diabetes [Kao et al].

EGCG also lowers blood pressure almost as effectively as the ACE-inhibitor drug, Enalapril, having significant implications for people with hypertension (high blood pressure) and cardiovascular disease [Kim et al].

Green tea catechins also have benefit for weight loss. A 2009 meta-analysis of 11 green tea catechin studies found that subjects consuming between 270 to 1200 mg green tea catechins / day (1 — 4 tsp of matcha powder per day) lost an average of 1.31 kg (~ 3 lbs) over 12 weeks with no other dietary or activity changes [Hursel].

Drinking 8-10 cups of green tea per day is enough to increase blood levels of EGCG into a measurably significant range [Kim et al], but matcha contains  137 times greater concentration of EGCG compared to green tip tea [Weiss et al].

WARNING TO PREGNANT WOMEN While EGCG has also been found to be similar in its effect to etoposide anddoxorubicin, a potent anti-cancer drug used in chemotherapy [Bandele et al], high intake of polyphenolic compounds during pregnancy is suspected to increase risk of neonatal leukemia. Bioflavonoid supplements (including green tea catechins) should not be used by pregnant women [Paolini et al].

Green Tea Shouldn’t Taste Bad!

The reason someone would find green tea has an “off flavor” was because the tea was either not fresh, not of a half-decent quality, was brewed at the wrong temperature or for the wrong length of time. Think about it this way; it all a person ever drank was cheap pre-ground coffee, they might think coffee tasted bad, too.

The fact is, one doesn’t need to order tea from Japan to enjoy a decent cup of green tea! I found the green teas below at a local Japanese grocery store and when brewed properly they are great as everyday tea.

If you aren’t adventurous to explore ethnic markets or time is limited, I can highly recommend the online supplier I mentioned above as having excellent price for the quality of green tea, very good explanations on their web page and quick delivery.

For everyday use, I have a little water cooler (yuzamashi) bowl and small single handed tea pot (kyuzu) so brewing a decent quality sencha green tea (my daily tea of choice) has become second nature, but as I mentioned above, one doesn’t need special equipment to make a decent cup of green tea!  All you need is the  right amount of fresh, good quality tea leaves steeped for the right length of time in hot water that’s at the right temperature. The only thing to keep in mind is that once the package of tea is opened, it needs to be stored in a sealed, airtight, light-proof container and used up within 3 months or sooner.

Making a good cup of green tea is not really much different than brewing a good cup of coffee. To make a good cup of coffee, one needs to consider the country / countries of origin of the beans, the bean roasting time and temperature, the brewing method involved (drip, espresso, French press, etc), the required water temperatures needed for that method, and the different grind of beans and a specific water-to-ground-bean ratio required for that brewing method. It sound’s complicated, but if you a few types of coffee regularly, it’s not hard.

It’s the same with green tea.

In one sense, there is a lot to learn at first to make a good cup of green tea but on the other hand, once you know a few basics and find a green tea or two you really enjoy, the rest is easy!

Tea has amazing health benefits, but unlike the cough medicine Buckley’s®, there is no need to drink tea that “tastes terrible, but it works”!

If you would like to know more about what I do as a Dietitian and how I can help you with weight loss or to seek to reverse the symptoms of metabolic syndrome, including Type 2 Diabetes, high blood pressure and other related markers, please send me a note using the Contact Me form on this web page.

If you would like to learn more about the services I offer and their costs, please click on the Service tab or have a look in the Shop.

To your good health!

Joy

You can follow me at:

       https://twitter.com/lchfRD

         https://www.facebook.com/BetterByDesignNutrition/

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Copyright ©2019 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

References

Gayathri Devi A, Henderson SA, Drewnowski A. Sensory acceptance of Japanese green tea and soy products is linked to genetic sensitivity to 6-n-propylthiouracil. Nutr Cancer. 1997;29(2):146-51

Hursel R, Viechtbauer W, Westerterp-Plantenga MS. The effects of green tea on weight loss and weight maintenance: a meta-analysis. Int J Obes (Lond) 2009;33:956—61.

Paolini, M, Sapone, A, Valgimigli, L, ”Avoidance of bioflavonoid supplements during pregnancy: a pathway to infant leukemia?”. Mutat Res 527 (1—2): 99—101. (Jun 2003)

Kao YH, Chang MJ, Chen CL, Tea, Obesity, and Diabetes, Molecular Nutrition & Food Research, 50 (2): 188—210, February 2006

Kim JA, Formoso G, Li Y, Potenza MA, Marasciulo FL, Montagnani M, Quon MJ., Epigallocatechin gallate, a green tea polyphenol, mediates NO-dependent vasodilation using signaling pathways in vascular endothelium requiring reactive oxygen species and Fyn, J Biol Chem. 2007 May 4;282(18):13736-45. Epub 2007 Mar 15.

Weiss, DJ, Anderton CR, Determination of catechins in matcha green tea by micellar electrokinetic chromatography, Journal of Chromatography A, Vol 1011(1—2):173-180, September 2003

Why I Posted My “Before” Pictures When I was Still Obese

INTRODUCTION: It is not uncommon for people to post their “before” pictures after they’ve reached their goal weight to show how much they’ve accomplished, but why on earth did I post pictures of myself when I was still obese and metabolically unwell? That’s a good question.

There’s a saying that “it is not the healthy who need a physician, but the sick” and while people will consult with Dietitian for many different reasons, those who are significantly overweight find it very difficult to take that first step when it is weight loss they’re seeking. Why?

People feel ashamed of being overweight or obese.

Oftentimes, overweight people feel that they are assumed to be undisciplined or lazy — that their condition is their own fault. They have heard over and over again that;

“If only they would eat less and move more they wouldn’t be so fat!”

or

“If only they ate ‘real food’ instead of ‘junk food’ they would be so much slimmer!”

Really?

If it were that simple, why would 1 in 4 Canadians (and 1 in 3 Americans) be obese?

Because it’s not that simple.

It’s been my experience that many overweight people and obese people often eat what has traditionally been thought of as a “healthy diet”; plenty of fruit and vegetables, low fat dairy products and only brown bread, rice and pasta and they feel frustrated and ashamed of being what is perceived as “a failure”.

Some have told me that sometimes their own healthcare providers have given them the impression that they must be being untruthful about what they’ve been eating because surely if they were eating the way they say, they would have been losing weight. In other words, they are not believed, or in stronger words, they are thought to be lying or at least incapable of accurately assessing how much they are ‘really’ eating.

Why would an overweight or obese person seek help in losing weight from a healthcare professional that views them as undisciplined, lazy or unrealistic about what they are eating?

They don’t.

Often people will try various diets that they read about online because no one will see them try and more importantly no one will see when they give up, feeling once again that they are ‘failures’.

I don’t think that overweight and obese people are failures. I believe many are doing what they’ve been told is the “right thing” but for different reasons. it is not working for them.  My role as a Dietitian is to help people understand what isn’t working and to enable them to be successful — without judgement.

It is for just such people that I posted my “fat” pictures before I ever started to lose weight!

I wanted people to see me as no different and certainly no better than they are, because I’m not. Sure, I have an undergraduate and graduate degree in nutrition, but I don’t get any “free passes” when it comes to losing weight and turning around my own metabolic health. I needed to do it just like everybody else.

I’ve lived each step of my weight loss and metabolic health recovery journey in public because I wanted people to experience in “real time” my frustrations and my victories. I wanted people to see that the path is not linear; that there are twists and turns and stalls, but yes it is possible to be successful. It just takes time and some dedicated work to get well and achieve a healthy body weight.

I look at it this way;

If it took me 20 years to become metabolically unhealthy and obese, what’s a couple of years to become metabolically healthy and normal weight?

Everyone’s weight loss and health restoration journey will be different.

There are no “magic bullets” or “super diets”— but there are different dietary and lifestyle options that can be pursued for success.

I can help.

 

If you would like to learn more about how I can help you or a family member achieve and maintain a healthy body weight and metabolic health, please send me a note using the Contact Me form located on the tab above.

To our good health!

Joy

You can follow me at:

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Copyright ©2019 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

Middle-Eastern Style Lentil Soup – whole food plant based

The new Canada Food Guide encourages a whole food plant-based diet which is a good option for those who are metabolically healthy — especially those who are insulin sensitive. The challenge is that I was diagnosed with Type 2 Diabetes 10 years ago and while I am in partial remission now as a result of dietary changes I implemented 23 months ago, on a cold winter day like yesterday I really wanted a bowl of my favourite homemade lentil soup.

I knew from testing my blood sugar in response to different foods that I was beginning to tolerate a small amount of whole, unground legumes such as chickpeas that had been soaked from the dried ones, then cooked. I also knew that leaving the lentils whole rather than pureeing them would reduce the blood sugar response and by adding additional non-starchy vegetables such as spinach and fresh green herbs would also help lower the glycemic response, so in the interest of science (of course) I decided to make the lentil soup and test my response two hours afterwards and the next morning.

The only significant source of carbohydrates that I ate yesterday was the soup which was ~20 g of carbs per bowl.  I was pleased and encouraged that after 23 months of changing how I ate that my blood glucose two hours after eating it was only 5.5 mmol/L (100 mg/dl), which was normal. This morning my fasting blood glucose was 6.3 mmol/L (114 mg/dl) which was significantly higher than what it has been the last few months eating a low carbohydrate diet, but considering the amount of slowly digestible carbohydrate in the soup, it was somewhat understandable.  To more accurately assess my glycemic response to the soup, I should have tested my blood sugar before I ate it, after 30 minutes, 60 minutes and 2 hours after eating it, as I did with my chickpea “experiment” as the 2 hour snapshot after 2 hours doesn’t provide any information as to what was happening to my blood glucose at 30 minutes and 60 minutes, which may have included a spike.

The soup was a nice treat and it was encouraging to me to continue to discover that as time goes on, I can reintroduce small amounts of whole-food carbohydrate sources without unduly impacting my blood sugars. Of course, being in remission from Type 2 Diabetes is not Diabetes  reversal, so I am by no means “cured”, but I am doing much better than 23 months ago.

As I know from several studies, including a 2015 study from Israel (Zeevi D, Korem T, Zmora N, et al. Personalized Nutrition by Prediction of Glycemic Responses. Cell. 2015 Nov 19;163(5):1079-1094), everyone’s glucose response to individual foods is different and the only way to know how each person will respond (whether Diabetic or non-diabetic / insulin resistant) is to test individual response to a specific amount of the food, which is what I did. While legumes are not something I would eat on a regular basis as it would negatively impact my glycated hemoglobin (HbA1C) level, it is certainly nice to be able to have it sometimes.

Of course, for those who are insulin sensitive, this is a delicious whole-food, largely plant based meal.

Below is the recipe for the soup. I included a piece of beef shank, but it can as easily be made without any meat for those that don’t eat it.

NOTE: This recipe is posted as a courtesy for those following a variety of different types of eating styles and not necessarily as part of a Meal Plan designed by me. This recipe may or may not be appropriate for you.

Middle Eastern Lentil Soup

Ingredients

1 medium yellow onion, chopped finely
1 medium carrot, diced
4 cloves fresh garlic, minced finely
2 tbsp olive oil
1 slice of beef shank, optional
2 cups small brown lentils, rinsed well
2 tsp coriander powder
1 tsp cumin powder
1/2 tsp freshly ground black pepper
kosher salt, to taste
1 cup fresh cilantro leaves (coriander greens), chopped
1 cup fresh parsley (flat leaf or curly), chopped
2 300 g packages of frozen chopped spinach, defrosted and squeezed dry
4 liters cold water

Herb Topping (optional)

3 green onions, minced finely
2 cloves fresh garlic, minced finely
1/2 cup fresh parsley, minced finely
1/2 cup fresh cilantro, minced finely
1 tbsp olive oil

Saute the green onions in the olive oil over a medium heat until wilted, but not browned, add the garlic and saute a minute or two then add the chopped parsley and cilantro and continue sauteing until the greens are slightly cooked.  Set aside to top each bowl of soup with, just before serving.

Method

  1. Saute the chopped onion in the olive oil until lightly browned
  2. Add the chopped carrot and saute until partially cooked
  3. Add the beef shank, if using and brown on both sides
  4. Add the minced garlic and saute (being careful not to let it brown as it would become bitter)
  5. Add the coriander and cumin powder, and keep stirring
  6. Toss in the rinsed brown lentils
  7. Season with salt and freshly ground black pepper
  8. Add cold water and stir to dislodge anything that may have stuck to the bottom
  9. Over a medium-low heat, bring to a simmer, skimming off any foam that accumulates from the meat protein
  10. Cook at medium-low for several hours, until the lentils are cooked but not too soft
  11. Twenty minutes before serving, add in the well-squeezed spinach, fresh parsley and fresh cilantro (coriander greens)
  12. Prepare the herb topping and set aside to top individual bowls of soupd when serving
  13. Enjoy!
Middle Eastern Style Lentil Soup

You can follow me at:

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Copyright ©2019 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

 

 

Finally Reaching Normal Body Weight – a Dietitian’s Journey

Me – May 2015

Today I reached “normal body weight” according to Body Mass Index (BMI) classification  no longer obese and not even overweight. Normal. It seems surreal.

When I began my health and weight loss journey on March 5, 2017 (19 1/2 months ago) I was obese. My weight bordered between Class I and Class II Obesity and I had multiple metabolic health issues. I was diagnosed with Type 2 Diabetes 10 years earlier, had elevated blood pressure and abnormal lipids (cholesterol).  Most significantly, I was in denial as to just how ill I really was. The undergraduate and post graduate degrees on my wall did not inform reality. The mirror did.

I didn’t feel well that day and took my blood pressure. It was dangerously high— classified as a hypertensive emergency. I decided to take my blood sugar too and it was way too high. I sat and considered the numbers of both and considered my options. At the time, I only saw two choices; I could go see my doctor who would have immediately put me on multiple medications or I could change my lifestyle. In hindsight the safest option would have been to do both, but I chose instead to begin to “practice what I teach”.

You see, I had two girlfriends suddenly die of natural causes within 3 months of each other just previous to that day; one of them I had known since high school and the other since university. They were both my age, both chose careers in healthcare, just like I did, and both died from preventable causes. They spent their lives helping others get well, yet unable to accomplish the same for themselves.  It was not for lack of trying, but for not having found a solution before death ended both of their lives. March 5, 2017, I realized that if I didn’t change I would likely die of heart attack or stroke, too. Their deaths may have saved my life.

I began a low carbohydrate diet immediately. I cut refined foods, ate whole unprocessed foods, didn’t avoid the fat that came with whole foods but didn’t add tons of fat either. While it helped a great deal, after several months I realized that I needed to lower my carbohydrates further in order to achieve the remission from Type 2 Diabetes that I sought.  I didn’t simply want to lose weight — I wanted to get healthy!

I consulted the experts and continued to make dietary modifications that got me closer to my goal. The first significant improvement was in blood pressure followed by blood sugar. I lost weight and more significantly lost inches off my waist.  While I hadn’t been formerly diagnosed with non-alcoholic fatty liver disease based on my lab work I more than likely had it. I tweaked and adjusted my Meal Plan many times over the last 19 1/2 months — each time moving myself closer and closer to my goal. Ten days ago I was within an inch of my waist circumference being half my height and now I am within 3/4 of an inch of it. It’s happening!

Body Mass Index (BMI) October 17 2018

Two days ago, I got on the scale and saw a series of digits that I had not seen since my twins were born 26 years ago tomorrow. I decided to crank some numbers.  I did a happy dance. I was almost there.  The photo on the left is weight category.

 

I am not one of those people that the press often writes about that pursued a low carbohydrate or ketogenic diet for “quick weight loss”.  I wanted to get well.  I chose a low carbohydrate diet for therapeutic reasons because it was my underlying high insulin levels which drove my high blood glucose and high blood pressure. To get well, I needed to address the cause, not the symptoms.

So here I am, having reached normal body weight!

Did I think at the beginning that I would actually get to this point? I wasn’t sure. I knew it was possible because I had helped others achieve it, but had never tried myself, so I didn’t know.

For health reasons, I no longer had the option of doing nothing!

At first, I set my preliminary goal as “no longer being obese“. Then I revised it to “being less overweight“.

I found some old photos recently of what I looked like as a young adult and realized what the weight was where I felt and looked my best then reset my goal weight once again. I knew it was entirely doable!

I am almost there!

Then the hard work begins.

Losing weight has been challenging, but not difficult.  Sure, I needed to determine what was holding things up at various stages of my journey and make dietary adjustments just as I do for my clients, but it’s much easier to do that for someone else than for oneself. The “hard work” will be finding out how to eat where I don’t lose any more weight, while maintaining my blood sugar and blood pressure at the best possible level.

If possible, I want to achieve full remission from Type 2 Diabetes and if not, I will learn how to maintain full reversal of symptoms.

I’ve documented the entire process throughout “A Dietitian’s Journey”, including “fat pictures” and lab test results to demonstrate the therapeutic benefit of a low carbohydrate diet and that this lifestyle is both practical and  sustainable.

Perhaps you would like to find out how I can help you achieve your own health and nutrition goals?

Please send me a note using the form on the Contact Me tab above and I’ll be happy to reply.

To your good health!

Joy

you can follow me at:

 https://twitter.com/lchfRD

  https://www.facebook.com/BetterByDesignNutrition/

Copyright ©2018 BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.

My Approach to Eating Low Carb and Links to Popular Recipes

Some people think there is only one type of “low carb” diet; one that includes lots of meat and lots of fat, but that’s not the case.  There are low carb diets that are higher in fat than protein, higher in protein than fat and those that are somewhat of a hybrid. The same thing is true when it comes to “ketogenic” or “keto” diets as it depends what it is being used for. A keto diet for epilepsy or as an adjunct treatment for Alzheimer’s disease or certain kinds of cancer will look very different than one used therapeutically to reverse metabolic conditions like Type 2 Diabetes. There isn’t a “one-size-fits-all” low carb or ketogenic (keto) diet.

As well, there aren’t any “one-size-fits-all” people! Some folks have higher protein needs because of their age or stage of life or the sports they engage and for people seeking weight loss those who have 15 or 20 pounds to lose won’t necessarily need to eat the same way as those with a great deal of weight to lose.

In addition, low carb diets often change over time. For example, those with a lot of weight to lose will often eat differently at the beginning of their weight-loss journey than they do towards the end of their weight loss because their body adapts and changes. As a result, these folks need to have their Meal Plan adjusted over time, whereas someone with a smaller amount of weight to lose may do fine with the same Meal Plan all the way through. Everybody’s different.

How I Approach It

My own meals usually center around some kind of grilled, roasted or stir-fried protein along with a generous serving of fresh low carb veggies plus some added healthy fat such as cold-pressed extra virgin olive oil or a touch of butter just to make things tasty. I don’t hesitate to sprinkle salads with pumpkin seeds or a few nuts, some berries and even a bit of crumbled goat cheese and drizzle it with olive or macadamia nut oil because this way I’m happy to eat a large bowl of it and it keeps me satisfied for hours. For those whose of my clients whose dietary needs are similar, I encourage them to do the same; switching up the type of nuts or seeds they use and changing the type of cold pressed oil they use, as each tastes very different. Even changing the type of vinaigrette from vinegar-based to lemon-based or using different types of vinegar or herbs adds more variety. There are so many kinds of meat, fish, poultry and vegetables that can be eaten and each can be prepared lots of different ways, so there’s no need to get bored eating the same thing.

Reversing Type 2 Diabetes

In a little over a year I’ve lost almost 40 pounds- first eating a low carb diet and then necessarily because of significant hyperinsulinemia (high levels of circulating insulin) and insulin resistance, a ketogenic diet. I’ve put my Type 2 Diabetes into remission while reversing my high cholesterol and high blood pressure and while I’ve not yet arrived at the point where my waist circumference is half my height (lowest risk) I am getting close.

Because I was Diabetic for 10 years and obese for longer than that, I tend to limit my own intake of low carb baked goods (muffins, pancakes and breads) that are often made from ground nuts or seeds and cheese as these are very  energy dense. I still have some of my own excess fat stores to lose as well as continuing to lose fat from places it should never have been in the first place (including very likely my liver) so eating extra dietary fat outside of those found naturally in whole, unprocessed foods (meat, fish, poultry, cheese, egg) doesn’t make much sense.

I do better with a low carb lower fat cauliflower crust pizza  (recipe below) or a low carb zucchini pizza crust (recipe coming soon!) over the very popular “fathead pizza” (based on almond flour and lots of fat from different kinds of cheese) or even my own Crisp Keto Pizza (recipe below) which is high in protein and fat but low in carbs. That’s why there are a few kinds of pizza recipes, so there’s a choice – not just for me, but my clients and visitors to my site. One can’t have too many healthy, tasty ways to eat pizza, right?

Most Popular Recipes

Below are a few of my most popular low carb recipes grouped by type of low carb diet. Please remember, not all recipes will be suitable for your specific health conditions or weight loss goals, so if in doubt please check with your Dietitian or physician. I hope you enjoy them.

Higher Fat Low Carb Recipes

For those that follow a high fat low carb lifestyle, below are a few of my most popular recipes. For me (and quite a few of my clients who are also in the weight loss phase) these are “sometimes foods” and not “everyday foods”.

Low Carb Beer-Batter Fish (seriously amazing)
Quiche Lorraine
Crisp Keto Pizza

Desserts in this category include my  Low Carb New York Cheesecake (amazingly good!) and Low Carb / Keto Ice Cream .

Low Carb Moderately High Fat

Recipes more suited to daily fare for me and those who are in the weight-loss phase of a low carb diet are posted here.  Some of the most popular are;
Crispy Cauliflower Pizza (lower in fat than the Crisp Keto Pizza above)
Low Carb Chow Mein
Low Carb Thai Green Curry
Spaghetti Zoodles with Bolognese Sauce
Low Carb Kaiser Buns great with sliced meat or cheese and lettuce (or used as a hamburger bun!).

This Low Carb Chocolate Chip Pancake recipe was recently posted but I’m pretty sure it will become a favourite, too. It is around my house!

Great everyday side dishes that can accompany a wide variety of poultry, fish, meat and veggies whether for the family or company are;
Low carb high protein broad noodles
Keto Yeast Rolls
Low Carb Roti (Indian flatbread)

Higher Fat Convenience Food Recipes

I have created and posted several recipes for higher fat protein bars if you need an easy, tasty and cost-efficient substitute for expensive low carb convenience bars on the market. These are;

Chocolate Orange Low Carb Protein Bars
Chocolate Mint Low Carb Protein Bars
Low Carb High Fat (Keto) Protein Bars

I even have a Low Carb Green Tea Matcha Smoothie that can be used to target abdominal fat in those following a higher fat low carb eating plan.

If you have questions about how I can help you to lose weight, reverse Type 2 Diabetes, high blood pressure or high cholesterol or to adopt a low-carb lifestyle for its other health benefits, please feel free to send me a note using the Contact Me form on this web page. I provide both in-person services in my Coquitlam (British Columbia) office, as well as services via Distance Consultation (phone or Skype) to those living elsewhere.

I hope you enjoy these recipes and please feel free to send me a message on social media (Facebook or Twitter, links below) if you have questions about any of the recipes or to post pictures when you make them.

To our good health!

Joy


you can follow me at:

 https://twitter.com/lchfRD

  https://www.facebook.com/BetterByDesignNutrition/

Copyright ©2018  BetterByDesign Nutrition Ltd.

LEGAL NOTICE: The contents of this blog, including text, images and cited statistics as well as all other material contained here (the ”content”) are for information purposes only.  The content is not intended to be a substitute for professional advice, medical diagnosis and/or treatment and is not suitable for self-administration without the knowledge of your physician and regular monitoring by your physician. Do not disregard medical advice and always consult your physician with any questions you may have regarding a medical condition or before implementing anything  you have read or heard in our content.